Author

E. Bartnicki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

K. M. Khanna· 3 shared A. H. Yeaton· 2 shared A. Herrera· 2 shared A. L. Moreira· 2 shared A. Rashidfarrokhi· 2 shared A. Tsirigos· 2 shared C. Loomis· 2 shared C. M. Rudin· 2 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

K. M. Khanna

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Tumor-intrinsic LKB1-LIF signaling axis establishes a myeloid niche to promote immune evasion and tumor growth

A. Rashidfarrokhi, R. Pillai, Y. Hao, W. Wu, B. Karadal-Ferrena, S. G. dimitriadoy, M. Cross, A. H. Yeaton, S. Ming Huang, A. J. Bhutkar, A. Herrera, S. Rajalingam, M. Hayashi, K.-l. Huang, E. Bartnicki, , T. Papagiannakopoulos

This study identifies a LKB1-LIF signaling axis in tumors that promotes immune evasion by reprogramming myeloid cells, and shows that inhibiting LIF signaling reduces tumor growth.

bioRxiv · Jul 15, 2023 · PreprintCancer Biology

MAVS signaling is required for clearance of chikungunya heart infection and prevention of chronic inflammation in vascular tissue

M. G. Noval, S. N. Spector, E. Bartnicki, F. Izzo, N. Narula, S. T. Yeung, P. Damani-Yokota, M. Z. Dewan, V. Mezzano, B. A. Rodriguez-Rodriguez, C. Loomis, K. M. Khanna, K. A. Stapleford

This study investigates CHIKV-induced heart disease using mouse models and human cells, showing MAVS signaling is critical for viral clearance and preventing chronic vascular inflammation.

bioRxiv · May 27, 2022 · PreprintMicrobiology

KEAP1 mutation in lung adenocarcinoma promotes immune evasion and immunotherapy resistance

A. M. Zavitsanou, Y. Hao, W. L. Wu, E. Bartnicki, R. Pillai, T. Karakousi, A. Herrera, A. Karatza, A. Rashidfarrokhi, S. Solis, M. Ciampricotti, A. H. Yeaton, E. Ivanova, C. A. Wohlhieter, T. B. Buus, , S. B. Koralov

This study shows KEAP1 mutations in lung adenocarcinoma lead to immune evasion by reducing dendritic cell and T cell responses, contributing to immunotherapy resistance.

bioRxiv · Sep 24, 2021 · PreprintCancer Biology