Author

I. Pallavicini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Research Focus:differential expression· 1

Frequent Co-Authors

S. Minucci· 3 shared R. Ravasio· 2 shared A. Di Martino· 1 shared A. O. Bigogno· 1 shared B. Duso· 1 shared C. B. Nava Lauson· 1 shared C. Gentile· 1 shared C. Steinkuhler· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersImmunology

S. Minucci

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivOct 27, 2023

LSD1 inhibition improves efficacy of adoptive T cell therapy by enhancing CD8+ T cell responsiveness.

I. Pallavicini, T. M. Frasconi, E. Ceccaci…·T. Manzo

This study shows that inhibiting LSD1 enhances CD8+ T cell function in adoptive T cell therapy, leading to improved antitumor immunity and synergistic effects with anti-PDL1 therapy.

DOI 10.1038/s41467-024-51500-9

mouseCITE-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintMay 2, 2021

Inhibiting LSD1 suppresses coronavirus-induced inflammation but spares innate antiviral activity

L. Mazzarella, F. Santoro, R. Ravasio…·P. G. Pelicci

The paper shows inhibiting LSD1 reduces coronavirus-induced inflammation while preserving innate antiviral activity in macrophages.

DOI 10.1101/2021.05.02.441948

mousebulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintFeb 16, 2021

Pharmacological inhibition of LSD1 triggers myeloid differentiation by targeting GSE1, a novel oncogene in AML

L. Nicosia, F. L. Boffo, E. Ceccacci…·T. Bonaldi

This study shows that LSD1 inhibitors induce AML cell differentiation by reducing GSE1, a novel oncogene, through transcriptional activation of immune response genes.

DOI 10.1101/2021.02.16.431315

humanbulk RNA-seq

𝕏 PostBluesky