Author

J. L. Cunningham

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:fungi (other)· 1 Organism:human· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. K. Bhandage· 1 shared B. Birnir· 1 shared B. Lubinski· 1 shared C. Ye· 1 shared D. Bugumbe· 1 shared D. G. Diel· 1 shared G. Whittaker· 1 shared J. A. Jaimes· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperPathology

A. K. Bhandage

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The SARS-CoV-2 Spike is a virulence determinant and plays a major role on the attenuated phenotype of Omicron virus in a feline model of infection

M. Martins, M. Nooruzzaman, J. L. Cunningham, C. Ye, L. C. Caserta, N. Jackson, L. Martinez-Sobrido, Y. Fang, D. G. Diel

This study shows the Omicron BA.1 Spike protein reduces SARS-CoV-2 virulence in a feline model by impairing viral spread and causing less severe disease.

bioRxiv · Oct 9, 2023Microbiology

Spike protein cleavage-activation mediated by the SARS-CoV-2 P681R mutation: a case-study from its first appearance in variant of interest (VOI) A.23.1 identified in Uganda

B. Lubinski, L. Frazier, M. Phan, D. Bugumbe, J. L. Cunningham, T. Tang, S. Daniel, M. Cotten, J. A. Jaimes, G. Whittaker

The paper analyzes the P681R mutation in the SARS-CoV-2 spike protein of variant A.23.1, showing increased furin cleavage and infectivity compared to earlier variants, but only when combined with other spike changes.

bioRxiv · Jun 30, 2021 · PreprintMicrobiology

Depression, GABA and age correlate with the plasma levels of inflammatory markers.

A. K. Bhandage, J. L. Cunningham, Z. Jin, Q. Shhen, S. Bongiovanni, S. V. Korol, M. Syk, M. Kamali-Moghaddam, L. Ekselius, B. Birnir

The paper examines correlations between depression, age, GABA levels, and plasma inflammatory markers, identifying potential peripheral biomarkers for depression.

bioRxiv · Jul 2, 2019Pathology