Author

J. L. Mitchell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:proteomics· 1

Frequent Co-Authors

A. P. Bye· 1 shared A. Pagliuzza· 1 shared B. Weston· 1 shared C. Dufour· 1 shared C. Sacdalan· 1 shared D. Hsu· 1 shared D. Rico· 1 shared E. Kroon· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperImmunology

A. P. Bye

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Release of Histone H3K4-reading transcription factors from chromosomes in mitosis is independent of adjacent H3 phosphorylation

R. J. Harris, M. Heer, M. D. Levasseur, T. N. Cartwright, B. Weston, J. L. Mitchell, J. M. Coxhead, L. Gaughan, L. Prendergast, D. Rico, J. M. G. Higgins

The paper investigates the interaction between H3K4me3 and H3T3ph during mitosis, finding that H3T3ph does not prevent H3K4 reader binding, challenging the histone code hypothesis in this context.

bioRxiv · Feb 28, 2023Cell Biology

HIV rapidly targets a diverse pool of CD4+ T cells to establish productive and latent infections

P. Gantner, S. Buranapraditkun, A. Pagliuzza, C. Dufour, M. Pardons, J. L. Mitchell, E. Kroon, C. Sacdalan, N. Tulmethakaan, S. Pinyakorn, M. L. Robb, N. Phanuphak, J. Ananworanich, D. Hsu, S. Vasan, , N. Chomont

This study identifies that HIV initially infects diverse CD4+ T cells in blood and lymph nodes, establishing both productive infections and latent reservoirs simultaneously.

bioRxiv · May 10, 2022Microbiology

Aberrant glycosylation of anti-SARS-CoV-2 IgG is a pro-thrombotic stimulus for platelets

A. P. Bye, W. Hoepel, J. L. Mitchell, S. Jegouic, S. Loureiro, T. Sage, S. de Taeye, M. van Gils, N. Kriek, N. Cooper, I. Jones, J. den Dunnen, J. M. Gibbins

The paper shows that aberrant glycosylation of anti-SARS-CoV-2 IgG enhances platelet activation and thrombosis via FcyRIIA, which can be inhibited by tyrosine kinase inhibitors and P2Y12 antagonists.

bioRxiv · Mar 26, 2021Immunology