Author

K. Heydari

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:gene regulatory network inference· 2 Approach:genetic perturbation / CRISPR· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:database / resource· 1

Frequent Co-Authors

A. Ashok· 1 shared A. Dillin· 1 shared A. J. Mathiowetz· 1 shared C. M. Butnaru· 1 shared D. E. Leto· 1 shared D. Hockemeyer· 1 shared D. Jullie· 1 shared D. W. Morgens· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCell Biology

A. Ashok

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Parallel CRISPR-Cas9 screens reveal mechanisms of PLIN2 and lipid droplet regulation

M. A. Roberts, K. K. Deol, M. Lange, D. E. Leto, A. J. Mathiowetz, J. Stevenson, S. H. Hashemi, D. W. Morgens, E. Easter, K. Heydari, M. A. Nalls, M. C. Bassik, M. Kampmann, R. R. Kopito, F. Faghri, J. A. Olzmann

The paper uses CRISPR-Cas9 screens to identify genes regulating PLIN2 and lipid droplet abundance, revealing new mechanisms and creating a resource for lipid metabolism research.

bioRxiv · Aug 27, 2022 · PreprintCell Biology

New factors for protein transport identified by a genome-wide CRISPRi screen in mammalian cells

L. Bassaganyas, S. J. Popa, M. Horlbeck, A. Ashok, S. E. Stewart, C. M. Butnaru, N. Brouwers, K. Heydari, J. Ripoche, J. Weissman, R. Schekman, V. Malhotra, K. Moreau, J. Villeneuve

The paper identifies two new factors, TTC17 and CCDC157, involved in protein transport through a CRISPRi screen in mammalian cells, showing their roles in Golgi structure and transport carrier fusion.

bioRxiv · Feb 21, 2019Cell Biology

Transient activation of the UPRER is an essential step in the acquisition of pluripotency during reprogramming

M. Simic, E. Moehle, R. Schinzel, F. Lorbeer, D. Jullie, J. Halloran, K. Heydari, M. Sanchez, D. Hockemeyer, A. Dillin

This study shows that transient activation of the UPRER is essential for successful reprogramming of somatic cells into pluripotency and can predict reprogramming efficiency.

bioRxiv · Nov 17, 2018Cell Biology