Author

K. Sha

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

J. J. Krolewski· 3 shared K. L. Nastiuk· 3 shared S. Singh· 3 shared A. Maolake· 2 shared C. Pan· 2 shared G. Chatta· 2 shared K. H. Eng· 2 shared R. Zhang· 2 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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3 shared papersCancer Biology

J. J. Krolewski

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Androgen deprivation triggers a cytokine signaling switch to induce immune suppression and prostate cancer recurrence

K. Sha, R. Zhang, A. Maolake, S. Singh, G. Chatta, K. H. Eng, K. L. Nastiuk, J. J. Krolewski

bioRxiv · Dec 1, 2023 · PreprintCancer Biology

TNF signaling is required for castration-induced vascular damage preceding prostate cancer regression

J. J. Krolewski, S. Singh, K. Sha, N. Jaiswal, S. G. Turowski, C. Pan, L. J. Rich, M. Seshadri, K. L. Nastiuk

This study shows TNF signaling is required for castration-induced vascular damage in prostate cancer, which contributes to tumor regression.

bioRxiv · Feb 5, 2022 · PreprintCancer Biology

MX2 viral substrate breadth and inhibitory activity are regulated by protein phosphorylation

G. Betancor, M. E. M. Bangham, J. K. Jeon, K. Sha, S. Lynham, J. M. Jimenez Guardeno, M. H. Malim

This paper identifies key phosphorylation sites in MX2 that regulate its antiviral activity and substrate specificity, showing that hypermorphic mutants can inhibit HIV-1 and resistant retroviruses.

bioRxiv · Jan 24, 2022Microbiology

Glucocorticoid signaling delays castration-induced regression in murine models of prostate cancer

A. Maolake, R. Zhang, K. Sha, S. Singh, C. Pan, B. Xu, G. Chatta, M. Mastri, K. H. Eng, J. J. Krolewski, K. L. Nastiuk

This study shows that glucocorticoid signaling delays castration-induced regression in murine prostate cancer models by maintaining tumor proliferation through GR activation, which can be blocked by the GR antagonist relacorilant.

bioRxiv · Oct 11, 2021 · PreprintCancer Biology