Author

M. Chi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Bharat· 1 shared A. C. McQuattie-Pimentel· 1 shared A. G. Robling· 1 shared A. Millan· 1 shared A. Mohamed· 1 shared A. V. Misharin· 1 shared A. Y. Meliton· 1 shared B. Chicana· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

A. Bharat

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3 papers

Recent·Most cited

The S2 subunit of spike encodes diverse targets for functional antibody responses to SARS-CoV-2

J. Guenthoer, M. E. Garrett, M. Lilly, D. M. Depierreux, F. Ruiz, M. Chi, C. I. Stoddard, V. Chohan, K. Sung, D. Ralph, H. Y. Chu, F. A. Matsen, J. Overbaugh

This paper identifies diverse functional antibodies targeting the S2 subunit of SARS-CoV-2 spike, including one with broad neutralization activity across multiple coronaviruses.

bioRxiv · Feb 26, 2024 · PreprintImmunology

Sclerostin depletion induces inflammation in the bone marrow of mice

C. Donham, B. Chicana, A. G. Robling, A. Mohamed, S. Elizaldi, M. Chi, B. Freeman, A. Millan, D. Murguesh, G. G. Loots, J. O. Manilay

This study shows that sclerostin depletion in mice leads to myeloid-biased hematopoiesis and increased inflammation in the bone marrow.

bioRxiv · Nov 1, 2020Immunology

The Aging Microenvironment Shapes Alveolar Macrophage Identity in Aging

A. C. McQuattie-Pimentel, Z. Ren, N. Joshi, S. Watanabe, T. Stoeger, M. Chi, Z. Lu, L. Sichizya, R. Piseaux, C.-I. Chen, S. Soberanes, P. A. Reyfman, J. M. Walter, K. R. Anekalla, J. M. Davis, , G. S. Budinger

This study shows that the aging lung microenvironment shapes alveolar macrophage identity, influencing their phenotype during aging and after injury.

bioRxiv · Jul 29, 2019 · PreprintImmunology