Author

M. Cicchini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

D. M. Feldser· 3 shared A. A. Gudiel· 1 shared A. C. Gladstein· 1 shared B. Stanger· 1 shared D. C. Brady· 1 shared F. Benavides· 1 shared G. P. Grothusen· 1 shared H. Mai· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersCancer Biology

D. M. Feldser

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3 papers

Recent·Most cited

p53 restoration in small cell lung cancer identifies a latent Cyclophilin-dependent necrosis mechanism

J. Acosta, Q. Li, N. F. Freebrug, N. Murali, G. P. Grothusen, M. Cicchini, H. Mai, A. C. Gladstein, K. M. Adler, K. R. Doerig, J. Li, M. Ruiz-Torres, K. L. Manning, B. Stanger, L. Busino, , D. M. Feldser

This study shows that p53 restoration in SCLC can induce necrosis via cyclophilin-dependent mechanisms, revealing subtype-specific responses and potential therapeutic targets.

bioRxiv · Jul 22, 2022Cancer Biology

Requirement for PKC epsilon in KRAS-driven lung tumorigenesis

R. Garg, M. Cooke, S. Wang, F. Benavides, M. C. Abba, M. Cicchini, D. M. Feldser, M. G. Kazanietz

This study shows PKCε is required for KRAS-driven lung tumorigenesis and identifies it as a potential therapeutic target.

bioRxiv · Jun 26, 2020 · PreprintCancer Biology

Copper is an essential regulator of the autophagic kinases ULK1/2 to drive lung adenocarcinoma

T. Tsang, J. M. Posimo, A. A. Gudiel, M. Cicchini, D. M. Feldser, D. C. Brady

This study identifies copper as a regulator of ULK1/2 kinase activity in lung adenocarcinoma, showing that copper chelation can inhibit autophagy and tumor growth.

bioRxiv · Oct 24, 2019Cancer Biology