Author

M. G. Lee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Aparicio· 1 shared A. G. Hoang· 1 shared A. K. Jain· 1 shared A. Lazar· 1 shared A. Raman· 1 shared A. Shah· 1 shared B. A. Foster· 1 shared B. Pradhan· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. Aparicio

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMay 14, 2022

KDM4A promotes the progression of neuroendocrine prostate cancer

C. S. L. Mak, M. Zhu, X. Liang…·G. Wang

This study identifies KDM4A as a key driver of neuroendocrine prostate cancer progression and shows that targeting KDM4A inhibits tumor growth in preclinical models.

DOI 10.1101/2022.05.14.491739

humanmouse

𝕏 PostBluesky

Cancer BiologybioRxivDec 27, 2018

Enhancer Reprogramming Confers Dependence on Glycolysis and IGF signaling in KMT2D Mutant Melanoma

M. Maitituoheti, E. Keung, M. Tang…·K. Rai

This study identifies KMT2D as a tumor suppressor in melanoma, showing its loss leads to metabolic reprogramming toward glycolysis and dependence on IGF signaling, suggesting therapeutic targeting of these pathways.

DOI 10.1016/j.celrep.2020.108293

mouseexperimental assay

𝕏 PostBluesky