Author

B. A. Foster

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:clustering· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2

Frequent Co-Authors

W. J. HUSS· 3 shared B. M. Gillard· 2 shared E. Karasik· 2 shared J. Zhang· 2 shared M. Mastri· 2 shared A. Aparicio· 1 shared A. G. Hoang· 1 shared A. K. Jain· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersCancer Biology

W. J. HUSS

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Author profile →

4 papers

Recent·Most cited

Syngeneic model of carcinogen-induced tumor mimics basal/squamous, stromal-rich, and neuroendocrine molecular and immunological features of muscle-invasive bladder cancer

S. D. Shah, B. M. Gillard, M. M. Wrobel, E. Karasik, M. T. Moser, M. Mastri, N. Sule, C. M. Brackett, W. J. HUSS, B. A. Foster

This paper establishes syngeneic bladder cancer tumor lines that mimic human molecular and immunological features of muscle-invasive bladder cancer subtypes and test cisplatin response.

Frontiers in Oncology · Feb 3, 2023Peer-reviewedCancer Biology

KDM4A promotes the progression of neuroendocrine prostate cancer

C. S. L. Mak, M. Zhu, X. Liang, F. Wang, F. Yuan, A. G. Hoang, X. Song, P. Shepherd, D. Liang, J. Suh, B. Pradhan, J. Park, M. Zhang, E. Metzger, R. Schule, , G. Wang

This study identifies KDM4A as a key driver of neuroendocrine prostate cancer progression and shows that targeting KDM4A inhibits tumor growth in preclinical models.

bioRxiv · May 14, 2022 · PreprintCancer Biology

Patient derived models of bladder cancer amplify tumor specific gene expression compared to surgical specimen while maintaining gene expression of molecular subtype and epithelial mesenchymal transition markers

M. Mastri, S. Ramakrishnan, S. Shah, E. Karasik, B. M. Gillard, M. M. Moser, B. K. Farmer, G. S. Chatta, A. Woloszynska, K. H. Eng, B. A. Foster, W. J. HUSS

This study compares gene expression profiles of patient-derived models (PDMs) with surgical specimens of bladder cancer, finding PDXs most similar to original tumors while maintaining EMT and molecular subtype markers.

bioRxiv · Feb 10, 2021 · PreprintCancer Biology

Ultra-deep sequencing differentiates patterns of skin clonal mutations associated with sun-exposure status and skin cancer burden

L. Wei, S. Christensen, M. Fitzgerald, J. Graham, N. Hutson, C. Zhang, Z. Huang, Q. Hu, F. Zhan, J. Xie, J. Zhang, S. Liu, E. Remenyik, E. Gellen, O. R. Colegio, , G. Paragh

This study uses ultra-deep sequencing to analyze clonal mutations in sun-exposed and non-sun-exposed skin, finding higher UV-signature mutations and mutation burden in sun-exposed samples.

bioRxiv · Jan 10, 2020 · PreprintCancer Biology