Author

M. Molin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Bioengineering· 1 Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:yeast· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 2 Assay:proteomics· 2 Research Focus:development & morphogenesis· 1 Assay:DNA methylation· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

C. Picazo· 2 shared A. B. Gjuvsland· 1 shared A. Vallet· 1 shared B. M. Burmann· 1 shared C. Asami· 1 shared C. Gao· 1 shared C. Gilchrist· 1 shared C. Gonzalez Pena· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBioengineering

C. Picazo

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Disulfide-bond-induced structural frustrationand dynamic disorder in a peroxiredoxin fromMAS NMR

L. Troussicot, A. Vallet, M. Molin, B. M. Burmann, P. Schanda

This study investigates how disulfide bonds affect the dynamics of peroxiredoxin assemblies using NMR, finding structural frustration and dynamic disorder.

bioRxiv · Feb 8, 2023Biophysics

The yeast eIF2 kinase Gcn2 facilitates H2O2-mediated feedback inhibition of both protein synthesis and ER oxidative folding during recombinant protein production

V. Gast, K. Campbell, C. Picazo, M. K. M. Engqvist, V. Siewers, M. Molin

This study shows that Gcn2 kinase mediates H2O2 feedback inhibition of protein synthesis and ER oxidative folding in yeast during recombinant protein production, and its removal enhances -amylase production.

bioRxiv · Feb 3, 2021Bioengineering

Genetically controlled mtDNA editing prevents ROS damage by arresting oxidative phosphorylation

S. Stenberg, J. Li, A. B. Gjuvsland, K. Persson, E. Demitz-Helin, C. Gonzalez Pena, J.-X. Yue, C. Gilchrist, T. Ärengard, P. Ghiaci, L. Larsson-Berglund, M. Zackrisson, J. L. Höög, M. Molin, G. Liti, , J. Warringer

The paper shows yeast cells use genetically controlled mtDNA editing to prevent ROS damage by shutting down oxidative phosphorylation.

bioRxiv · Nov 20, 2020 · PreprintCell Biology

Peroxiredoxin promotes longevity and H2O2-resistance in yeast through redox-modulation of PKA

F. Roger, C. Picazo, W. Reiter, M. Libiad, C. Asami, S. Hanzen, C. Gao, G. Lagniel, N. Welkenhuysen, J. Labarre, T. Nyström, M. Grotli, M. Hartl, M. B. Toledano, M. Molin

This study shows that Tsa1 promotes longevity and H2O2 resistance in yeast by modulating the PKA pathway through redox modifications.

bioRxiv · Jun 23, 2020Biochemistry