Author

N. Anthony

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:drug response / drug discovery· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Adam· 1 shared A. Arakelyan· 1 shared B. Dwivedi· 1 shared B. N. Vardarajan· 1 shared B. Rackley· 1 shared C. P. Decicco· 1 shared C.-S. Seong· 1 shared D. Huang· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperCancer Biology

A. Adam

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Author profile →

3 papers

Recent·Most cited

Pharmacologic inhibition of BAF chromatin remodeling complexes as a therapeutic approach to transcription factor-dependent cancers

R. C. Centore, L. M. M. Soares, S. Topal, R. G. Vaswani, K. Ichikawa, Z. Li, H. Fan, J. W. Setser, D. L. Lahr, L. E. Zawadzke, X. Chen, K. D. Barnash, J. Muwanguzi, N. Anthony, G. J. Sandoval, , S. F. Bellon

This study identifies and characterizes inhibitors of SMARCA4/SMARCA2 ATPases, showing their ability to induce lineage-specific chromatin changes and tumor regression in uveal melanoma.

bioRxiv · Sep 11, 2023 · PreprintCancer Biology

Retrotransposons facilitate tissue-specific horizontal Transfer of circulating tumor DNA

M. cinar, L. Martinez-Medina, P. K. Puvvula, A. Arakelyan, B. N. Vardarajan, N. Anthony, G. P. Nagaraju, D. Park, L. Feng, F. Sheff, M. Mosunjac, D. Saxe, S. Flygare, O. B. Alese, J. Kaufman, , L. Bernal-Mizrachi

This study shows retrotransposons enable tissue-specific horizontal gene transfer of circulating tumor DNA in cancer cells, influencing treatment response and opening new therapeutic avenues.

bioRxiv · Aug 10, 2022 · PreprintCancer Biology

The level of oncogenic Ras controls the malignant transformation of Lkb1 mutant tissue in vivo.

B. Rackley, C.-S. Seong, E. Kiely, R. E. Parker, M. Rupji, B. Dwivedi, J. M. Heddleston, W. Giang, N. Anthony, T.-L. Chew, M. Gilbert-Ross

A Drosophila model shows that high oncogenic Ras levels drive malignant transformation in Lkb1 mutant tissue, with implications for human lung adenocarcinoma.

bioRxiv · Sep 25, 2020Cancer Biology