Author

N. Kalogriopoulos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

I. Lopez-Sanchez· 2 shared P. Ghosh· 2 shared S. Roy· 2 shared C. Lin· 1 shared C. Rohena· 1 shared D. Sahoo· 1 shared F. Murray· 1 shared I. Kufareva· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersBiochemistry

I. Lopez-Sanchez

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2 papers

Recent·Most cited

Receptor tyrosine kinases activate heterotrimeric G proteins via phosphorylation within the interdomain cleft of Gαi

N. Kalogriopoulos, I. Lopez-Sanchez, C. Lin, T. Ngo, K. Midde, S. Roy, N. Aznar, F. Murray, M. Garcia-Marcos, I. Kufareva, M. Ghassemian, P. Ghosh

This study identifies how receptor tyrosine kinases activate heterotrimeric G proteins through phosphorylation of Gi's interdomain cleft tyrosines, revealing a tyrosine-based signaling mechanism with implications in cancer.

bioRxiv · Aug 9, 2020Biochemistry

GIV-Kindlin interaction is required for Kindlin-Mediated Integrin Recognition and Activation

C. Rohena, N. Kalogriopoulos, N. Rajapakse, S. Roy, I. Lopez-Sanchez, J. Ablack, D. Sahoo, P. Ghosh

This paper shows that GIV binds Kindlin-2 to enhance integrin activation, promoting cell adhesion and invasion, and links this interaction to metastasis in tumor transcriptomics.

bioRxiv · Dec 10, 2019Cell Biology