Author

N. Visser

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Organism:mouse· 2 Assay:scRNA-seq· 2

Frequent Co-Authors

E. Ammatuna· 2 shared G. Huls· 2 shared A. Diepstra· 1 shared A. Hodgins-Davis· 1 shared A. James· 1 shared A. K. Huber· 1 shared A. Lucena-Araujo· 1 shared A. M. Weber· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

E. Ammatuna

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Galectin-9 has non-apoptotic cytotoxic activity towards Acute Myeloid Leukemia independent of cytarabine resistance

G. Choukrani, N. Visser, N. Ustyanovska Avtenyuk, M. Olthuis, G. Marsman, E. Ammatuna, H. J. Lourens, T. Niki, G. Huls, E. Bremer, V. R. Wiersma

This study shows Galectin-9 induces non-apoptotic cytotoxicity in AML cells, including resistant ones, and enhances the effect of Azacytidine.

bioRxiv · Jan 12, 2023Cancer Biology

Human commensal Candida albicans strains demonstrate substantial within-host diversity and retained pathogenic potential

F. M. Anderson, N. Visser, K. Amses, A. Hodgins-Davis, A. M. Weber, K. M. Metzner, M. J. McFadden, R. E. Mills, M. J. O'Meara, T. Y. James, T. R. OMeara

The paper examines genetic and phenotypic diversity in commensal Candida albicans strains from healthy donors and shows they retain pathogenic potential.

bioRxiv · Sep 9, 2022 · PreprintMicrobiology

M2-polarized macrophages control LSC fate by enhancing stemness, homing, immune evasion and metabolic reprogramming

I. Weinhauser, D. Pereira-Martins, L. Almeida, J. Hilberink, C. Ortiz, D. Silveira, L. Quek, C. Araujo, T. Bianco, A. Lucena-Araujo, N. Visser, S. Hogeling, A. Diepstra, E. Ammatuna, G. Huls, , J. J. Schuringa

The paper shows M2-polarized macrophages promote leukemia stem cell survival by enhancing stemness, homing, immune evasion, and metabolic reprogramming.

bioRxiv · May 6, 2022Cancer Biology

Regulation of heterotopic ossification through local inflammatory monocytes in a mouse model of aberrant wound healing

A. K. Huber, M. Sorkin, C. Hwang, W. F. Carson, R. Menon, J. Li, K. Vasquez, C. Pagani, N. Patel, S. Li, N. Visser, Y. Niknafs, S. Loder, M. Scola, D. Nycz, , B. Levi

This study identifies TGFβ1-producing monocytes/macrophages as drivers of heterotopic ossification in a mouse model and shows CD47 activation can reduce HO by modulating these cells.

bioRxiv · Dec 11, 2019 · PreprintImmunology