Author

C. Hwang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1

Frequent Co-Authors

J. Li· 2 shared A. Iskander· 1 shared A. James· 1 shared A. K. Huber· 1 shared B. Levi· 1 shared B. V. Chakravarthi· 1 shared C. Pagani· 1 shared C. Rogers· 1 shared

Activity by Year

2019-2019 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

J. Li

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Regulation of heterotopic ossification through local inflammatory monocytes in a mouse model of aberrant wound healing

A. K. Huber, M. Sorkin, C. Hwang, W. F. Carson, R. Menon, J. Li, K. Vasquez, C. Pagani, N. Patel, S. Li, N. Visser, Y. Niknafs, S. Loder, M. Scola, D. Nycz, , B. Levi

This study identifies TGFβ1-producing monocytes/macrophages as drivers of heterotopic ossification in a mouse model and shows CD47 activation can reduce HO by modulating these cells.

bioRxiv · Dec 11, 2019 · PreprintImmunology

Pseudogene associated recurrent gene fusion in prostate cancer

B. V. Chakravarthi, P. D. Arachchige, S. Carskadon, S. Kalyana-Sundaram, J. Li, K.-H. H. Wu, D. S. Chandrashekar, J. O. Peabody, H. Stricker, C. Hwang, D. A. Chitale, S. R. Williamson, N. S. Gupta, N. M. Navone, C. Rogers, , N. Palanisamy

The paper identifies and validates a novel gene fusion in prostate cancer involving KLK4 and KLKP1, showing its association with high Gleason grade tumors and potential as a biomarker.

bioRxiv · Jun 11, 2019Cancer Biology

Castration-resistant prostate cancer: Androgen receptor inactivation induces telomere DNA damage, and damage response inhibition leads to cell death

V. Reddy, A. Iskander, C. Hwang, G. Divine, M. Menon, E. R. Barrack, G. P.-v. Reddy, S.-H. Kim

This study shows that combining an AR antagonist with an ATM inhibitor induces telomere DNA damage and DDR inhibition, leading to cell death in CRPC cells.

bioRxiv · Jan 9, 2019Cancer Biology