Author

P. Germon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 6 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 7 Approach:experimental assay· 6 Organism:chicken / avian· 4 Assay:bulk RNA-seq· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2

Frequent Co-Authors

A. Remot· 5 shared N. Winter· 4 shared E. Doz-Deblauwe· 3 shared F. Carreras· 3 shared M. Rambault· 3 shared F. B. Gilbert· 2 shared P. Cunha· 2 shared P. Rainard· 2 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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5 shared papersImmunology

A. Remot

Mutual co-authors in cluster

Author profile →

7 papers

Recent·Most cited

Neutrophil subsets play dual roles in tuberculosis by producing inflammasome dependent-IL-1β or suppressing T-cells via PD-L1

E. Doz-Deblauwe, B. Bounab, F. Carreras, J. Silverira-Fahel, S. C. Oliveira, M. Lamkanfi, Y. Le Vern, P. GERMON, J. Pichon, F. Kempf, C. Paget, A. Remot, N. Winter

The paper identifies two neutrophil subsets in tuberculosis with opposing roles: one promoting inflammation via IL-1β and another suppressing T-cells via PD-L1.

bioRxiv · Nov 17, 2023 · PreprintImmunology

Bovine blood and milk T-cell subsets in distinct states of activation and differentiation during subclinical Staphylococcus aureus mastitis

N. Tucker, P. Cunha, F. B. Gilbert, M. Rambault, K. R. Santos, A. Remot, P. Germon, P. Rainard, R. P. Martins

This study identifies changes in T-cell subsets in bovine blood and milk during subclinical Staphylococcus aureus mastitis, showing activation of CD4+, CD8+, and γδ T-cells and differences in naive and memory T-cell proportions.

bioRxiv · Jan 12, 2023 · PreprintImmunology

MHC-II expressing neutrophils circulate in blood and milk during mastitis and show high microbicidal activity

M. Rambault, F. B. Gilbert, P. Roussel, A. Tessier, V. David, P. Germon, N. Winter, A. Remot

This study identifies MHC-II expressing neutrophils in blood and milk during mastitis, showing higher microbicidal activity and correlation with T lymphocytes in subclinical cases.

bioRxiv · Sep 1, 2022 · PreprintImmunology

Mycobacterial infection of precision cut lung slices reveals that the type 1 interferon pathway is locally induced by Mycobacterium bovis but not M. tuberculosis in different cattle breeds

A. Remot, F. Carreras, A. Coupe, E. Doz-Deblauwe, M.-L. Boschiroli, J. A. Browne, Q. Marquant, D. Descamps, F. Archer, A. Aseffa, P. Germon, S. V. Gordon, N. Winter

The paper uses precision-cut lung slices to compare immune responses to Mycobacterium bovis and M. tuberculosis in two cattle breeds, finding that M. bovis induces the type I interferon pathway while M. tuberculosis does not.

bioRxiv · Apr 16, 2021Immunology

LPS core type diversity in the Escherichia coli species and associations with phylogeny and virulence gene repertoire.

S. O. Leclercq, M. Branger, D. G. Smith, P. GERMON

The paper analyzes the distribution of LPS core types in E. coli phylogroups and their association with virulence gene repertoires using whole genome sequences.

bioRxiv · Mar 22, 2021 · PreprintMicrobiology

Neutrophils encompass a regulatory subset suppressing T cells in apparently healthy cattle and mice

M. Rambault, E. Doz-Deblauwe, Y. Le Vern, F. Carreras, P. Cunha, P. Germon, P. Rainard, N. Winter, A. Remot

The paper identifies a regulatory subset of neutrophils in mice and cattle that suppress T cell proliferation through MHC-II expression and contact-dependent mechanisms.

bioRxiv · Nov 2, 2020Immunology

Leptospiral LPS escapes mouse TLR4 internalization and TRIF?associated antimicrobial responses through O antigen and associated lipoproteins

D. Bonhomme, I. Santecchia, F. Vernel-Pauillac, M. Caroff, P. Germon, G. Murray, B. Adler, I. G. Boneca, C. Werts

This study shows leptospiral LPS avoids triggering the TRIF-dependent antimicrobial response in mice by not internalizing TLR4, and this evasion depends on O antigen and co-purifying lipoproteins.

bioRxiv · May 18, 2020Immunology