Author

R. Bucki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. E. Patteson· 2 shared L. Suprewicz· 2 shared A. Nicolas-Boluda· 1 shared A. Wilczewska· 1 shared A. Wolfe· 1 shared C. Kantari-Mimoun· 1 shared D. A. Germann· 1 shared D. Iwamoto· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersBiophysics

A. E. Patteson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Extracellular vimentin is sufficient to promote cell attachment, spreading, and motility by a mechanism involving N-acetyl glucosamine-containing structures

R. Bucki, D. Iwamoto, X. Shi, K. E. Kerr, F. J. Byfield, L. Suprewicz, K. Sklodowski, J. Sutaria, P. Misiak, A. Wilczewska, S. Ramachandran, A. Wolfe, M.-T. H. Thanh, E. Whalen, A. E. Patteson, P. Janmey

This study shows extracellular vimentin promotes cell attachment, spreading, and motility via N-acetyl glucosamine-containing structures, identifying a novel adhesion mechanism.

bioRxiv · Nov 28, 2022Biophysics

Extracellular vimentin as a target against SARS- CoV-2 host cell invasion

L. Suprewicz, M. Swoger, S. Gupta, E. Piktel, F. F. Byfield, D. V. Iwamoto, D. A. Germann, J. Reszec, N. Marcinczyk, R. Carroll, M. Lenart, K. Pyrc, P. A. Janmey, J. M. Schwarz, R. Bucki, A. E. Patteson

The paper identifies extracellular vimentin as a potential target for preventing SARS-CoV-2 infection by showing it facilitates viral entry through interaction with the spike protein and ACE2.

bioRxiv · Jan 8, 2021Biophysics

Tumor stiffening reversion through collagen crosslinking inhibition improves T cell migration and anti-PD-1 treatment

A. Nicolas-Boluda, J. Vaquero, S. Barrin, C. Kantari-Mimoun, M. Ponzo, G. Renault, P. Deptula, K. Pogoda, R. Bucki, I. Cascone, J. Courty, L. Fouassier, F. Gazeau, E. Donnadieu

This study shows that inhibiting collagen crosslinking reduces tumor stiffness, enhances T cell migration, and improves anti-PD-1 treatment efficacy in mouse tumor models.

bioRxiv · May 19, 2020Cancer Biology