Author

R. Peter

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:deep learning· 1 Organism:human· 1 Assay:imaging· 1 Approach:machine learning (classical)· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

A. P. Bidkar· 1 shared A. Wadhwa· 1 shared A.-M. Donachie· 1 shared B. Liu· 1 shared C. V. Pastuskovas· 1 shared C. Y. Chabu· 1 shared D. Beckford-Vera· 1 shared D. M. Wilson· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperCancer Biology

A. P. Bidkar

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Author profile →

3 papers

Recent·Most cited

A Light sheet fluorescence microscopy and machine learning-based approach to investigate drug and biomarker distribution in whole organs and tumors.

N. Kumar, P. Hrobar, M. Vagenknecht, J. Soukup, N. Patterson, P. Bloomingdale, T. Freshwater, S. Bardehle, R. Peter, R. Mangadu, C. V. Pastuskovas, C. Y. Chabu, M. T. Cancilla

This paper uses light sheet fluorescence microscopy and machine learning to study drug and biomarker distribution in whole organs and tumors, showing improved segmentation accuracy and subcellular resolution.

bioRxiv · Sep 16, 2023 · PreprintCancer Biology

Treatment of prostate cancer with CD46 targeted 225Ac alpha particle radioimmunotherapy

A. P. Bidkar, S. Wang, K. N. Bobba, E. Chan, S. Bidlingmaier, E. A. Egusa, R. Peter, U. Ali, N. Meher, A. Wadhwa, S. Dhrona, D. Beckford-Vera, Y. Su, R. Tang, L. Zhang, , R. R. Flavell

This study develops and evaluates [225Ac]DOTA-YS5, a radioimmunotherapy for prostate cancer, showing high efficacy in xenograft models but nephrotoxicity at higher doses.

bioRxiv · Oct 13, 2022 · PreprintCancer Biology

Diminazene resistance in Trypanosoma congolense is linked to reduced mitochondrial membrane potential and not to reduced transport capacity

H. De Koning, L. V. Carruthers, J. C. Munday, G. U. Ebiloma, P. Steketee, S. Jayaraman, G. D. Campagnaro, M. A. Ungogo, A.-M. Donachie, T. G. Rowan, R. Peter, L. J. Morrison, M. P. Barrett

This study investigates diminazene resistance in Trypanosoma congolense and finds that resistance is due to reduced mitochondrial membrane potential rather than reduced drug transport capacity.

bioRxiv · Jul 28, 2020Microbiology