Author

R.-X. Lin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

M. Donowitz· 2 shared R. Sarker· 2 shared V. Singh· 2 shared C. M. Tse· 1 shared H. Yang· 1 shared H. Yu· 1 shared J. F. Abel· 1 shared J. In· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersPhysiology

M. Donowitz

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SLC26A3 (DRA) is stimulated in a synergistic, intracellular Ca2+ dependent manner by cAMP AND ATP in intestinal epithelial cells

R. Sarker, R.-X. Lin, V. Singh, M. Donowitz, C. M. Tse

The paper shows that forskolin and ATP synergistically increase DRA activity in intestinal epithelial cells through intracellular Ca2+ elevation, which is blocked by BAPTA-AM.

bioRxiv · Dec 2, 2022Physiology

CHRONIC INFLAMMATION IN ULCERATIVE COLITIS CAUSES LONG TERM CHANGES IN GOBLETCELL FUNCTION

V. Singh, K. Johnson, J. Yin, S. Lee, R.-X. Lin, H. Yu, J. In, J. F. Abel, N. Zachos

This study shows that UC colonoids have persistent defects in mucus secretion despite reduced GC numbers, suggesting long-term epithelial dysfunction contributes to UC recurrence.

bioRxiv · Mar 17, 2021Physiology

Chronic exposure of ErbB TKIs inhibits DRA expression and activity through an ERK/Elk-1/CREB/AP-1 dependent pathway

H. Yang, R.-X. Lin, R. Sarker, M. Donowitz

bioRxiv · Oct 16, 2020 · PreprintPharmacology & Toxicology