Author

S. Balk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

M. Brown· 2 shared A. Boosma-Moody· 1 shared A. K. Tewari· 1 shared A. Sowalsky· 1 shared A. T. M. Cheung· 1 shared A. Varkaris· 1 shared B. Ersoy-Fazlioglu· 1 shared D. G. Livitz· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

M. Brown

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Increased chromatin accessibility mediated by nuclear factor I drives transition to androgen receptor splice variant dependence in castration-resistant prostate cancer

L. Poluben, M. Nouri, J. Liang, A. Varkaris, B. Ersoy-Fazlioglu, O. Voznesensky, I. Lee, X. Qiu, L. Cato, J.-H. Seo, M. Freedman, A. Sowalsky, N. Lack, E. Corey, P. Nelson, , S. Balk

This study shows that ARv7 drives AR activity in castration-resistant prostate cancer through increased chromatin accessibility mediated by NFI factors.

bioRxiv · Jan 10, 2024 · PreprintCancer Biology

Molecular features of exceptional response to neoadjuvant anti-androgen therapy in high-risk localized prostate cancer

A. K. Tewari, A. T. M. Cheung, J. Crowdis, J. R. Conway, S. Y. Camp, S. A. Wankowicz, D. G. Livitz, J. Park, R. T. Lis, A. Boosma-Moody, M. X. He, S. H. AlDubayan, Z. Zhang, R. R. McKay, I. Leshchiner, , E. Van Allen

bioRxiv · Apr 20, 2021Cancer Biology

Ubiquitin Ligase MARCH5 Regulates Apoptosis through Mediation of Stress-Induced and NOXA-Dependent MCL1 Degradation

S. Arai, S. Chen, L. Xie, S. Balk

This study identifies MARCH5 as a mediator of NOXA-dependent MCL1 degradation in prostate cancer, enhancing apoptosis when combined with BH3 mimetics.

bioRxiv · Jan 12, 2020 · PreprintCancer Biology