Author

V. Mournetas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Cancer Biology· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 5 Assay:bulk RNA-seq· 4 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 3 Approach:statistical model· 3 Research Focus:differential expression· 2 Organism:mouse· 2

Frequent Co-Authors

C. Pinset· 3 shared E. Massourides· 2 shared A. Boman· 1 shared A. Bourdon· 1 shared A. Corbin· 1 shared A. Dutour· 1 shared A. Marnef· 1 shared A. Occhipinti· 1 shared

Activity by Year

2017-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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3 shared papersPathology

C. Pinset

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Dystrophin deficiency impairs cell junction formation during embryonic myogenesis

E. Mozin, E. Massourides, V. Mournetas, C. Lievre, A. Bourdon, D. L. Jackson, J. S. Packer, J. Seong, C. Trapnell, C. Le Guiner, O. Adjali, C. Pinset, D. L. Mack, J.-B. Dupont

This study shows that dystrophin deficiency disrupts cell junction formation during embryonic myogenesis in human pluripotent stem cells.

bioRxiv · Dec 5, 2023 · PreprintPathology

Fusion-negative Rhabdomyosarcoma 3D-organoids as an innovative model to predict resistance to cell death inducers

C. Savary, P. Huchede, L. Luciana, A. Tourbez, C. Deligne, C. Picard, T. Diot, C. Coquet, N. Meynard, M. Le Grand, L. Tonon, N. Gadot, C. Degletagne, S. Leon, V. Attignon, , L. Broutier

The paper develops a 3D-organoid model of fusion-negative rhabdomyosarcoma to predict resistance to cell death inducers using omics data.

bioRxiv · Sep 6, 2022 · PreprintCancer Biology

Gene co-expression network analysis of the human gut commensal bacterium Faecalibacterium prausnitzii based on WGCNA in R-Shiny

S. Auger, V. Mournetas, H. Chiapello, V. Loux, P. Langella, J.-M. Chatel

This study uses WGCNA to analyze gene expression in F. prausnitzii, identifying co-expression modules linked to metabolic processes and stress response mechanisms.

bioRxiv · Jul 10, 2022Microbiology

Loss of full-length dystrophin expression results in major cell-autonomous abnormalities in proliferating myoblasts

M. R. Gosselin, V. Mournetas, M. Borczyk, S. Verma, A. Occhipinti, J. Rog, L. Bozycki, M. Korostynski, S. Robson, C. Angione, C. Pinset, D. C. Gorecki

This study shows that loss of full-length dystrophin in myoblasts causes cell-autonomous defects in gene expression, proliferation, chemotaxis, and differentiation, contributing to muscle regeneration failure in Duchenne muscular dystrophy.

bioRxiv · Aug 24, 2021Pathology

Efficient genome editing in primary cells and in vivo using viral-derived "Nanoblades" loaded with Cas9/sgRNA ribonucleoproteins

P. E. Mangeot, V. Rissons, F. Fusil, A. Marnef, E. Laurent, J. Blin, V. Mournetas, E. Massourides, T. J. M. Sohier, A. Corbin, F. Aube, C. Pinset, L. Schaeffer, G. Legube, F.-L. Cosset, , E. P. Ricci

bioRxiv · Oct 12, 2017Cell Biology