Mohd Suhail Rizvi

The paper uses an active vertex model to show that spatial velocity correlations can distinguish four modes of epithelial activity based on their spatial organization and dynamics.

This paper identifies SARS-CoV-2 PLpro as a driver of epithelial stress and tissue damage across species by activating metabolic and inflammatory pathways in Drosophila and mammalian models.

This study proposes a biomechanical mechanism for collagen architecture in articular cartilage, showing how fluid flow from joint movements guides fiber alignment and adapts to activity and disease.

This study uses a vertex-based model to show how cell motility affects tissue viscoelasticity and morphological responses to mechanical and biochemical cues in the presomitic mesoderm.

This study develops a mathematical model of ATP metabolism in chondrocytes to show that chronic hypoxia shifts metabolism to glycolysis, reducing ATP and causing ECM loss, with therapeutic success requiring metabolic and structural interventions.

This study introduces a new method to measure energy dissipation during cell-cell junction rupture and shows that cortical tension and shape relaxation time determine whether junctions behave ductile or brittle.

The paper develops a minimal model to study how planar cell polarity emerges from local interactions and global gradients, showing that gradients enhance robustness and directionality.

The paper shows that degenerating larval epidermal cells generate pull forces that drive thorax closure in Drosophila by contracting and shrinking, facilitating HE migration and zipping.

The paper demonstrates a speed-detachment tradeoff in motor proteins and shows how this relationship follows a power law, affecting processivity and transport efficiency.