Marcus A. Garcia

This study shows that micro- and nanoplastics disrupt human brain development and function by inducing inflammation, cellular senescence, and altered electrophysiological activity.

The paper identifies synthetic plastics in human brain microvascular deposits using fluorescence and chemical analysis, suggesting potential links to microvascular pathology.

This study examines metabolomic, lipidomic, and enterohormone changes during progression from MASLD to MASH in mice, identifying cholic acid elevation and altered enteroendocrine hormone levels.

This study finds that smoking increases microplastic (MNP) and cadmium (Cd) levels in lung tissues, with a strong correlation between PE microplastics and Cd accumulation.

This study shows that microplastic exposure alters gut microbiome composition and serum metabolome in mice, with sex-specific and dose-dependent effects.

This study shows chronic cadmium exposure combined with hyperglycemia worsens mitochondrial dysfunction in liver cells, suggesting a role in MASLD development.

This study examines the neurometabolomic effects of wood smoke exposure in aged mice and evaluates the protective effects of anti-aging therapies.

The paper investigates the presence of microplastics in brain regions with white matter hyperintensities using post-mortem MRI, histopathology, and pyrGC/MS, suggesting a potential link between microplastics and vascular injury in cognitive impairment.