Author

Sandrine Horman

UCLouvainORCID 0000-0003-3031-2075Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Alice MarinoiD· 3 shared Christophe BeauloyeiD· 3 shared Jérôme AmbroiseiD· 3 shared Luc BertrandiD· 3 shared Audrey Ginion· 2 shared Caroline BouziniD· 2 shared Laura Guilbert· 2 shared Andreas Unger· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersPathology

Alice MarinoORCID

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3 papers

Recent·Most cited

PathologybioRxivPreprintJul 6, 2026

Platelet GARP-dependent activation of TGF-β1 limits inflammation and promotes cardiac repair after myocardial infarction

Cécile Dufeys, Julie Bodart, Audrey Ginion…·Sandrine Horman

This study shows that platelet GARP activates TGF-β1 to limit inflammation and promote cardiac repair after myocardial infarction.

DOI 10.64898/2026.07.01.735778

mouseexperimental assay

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Cell BiologyCardiovascular ResearchPeer-reviewedJan 30, 2026

Sodium-myo-inositol cotransporter-1, SMIT1, promotes cardiac hypertrophy and fibrosis induced by pressure overload in mice

Alice Marino, Julien Cumps, Laura Guilbert…·Christophe Beauloye

This study shows that SMIT1 promotes cardiac hypertrophy and fibrosis in mice under pressure overload by enhancing IP3/Ca2+-dependent signaling pathways.

3 citationstop 2%

mousebulk RNA-seq

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PhysiologybioRxivPreprintOct 23, 2025

skNAC is a Key Driver of Cardiomyocyte Integrity Against Pathological Cardiac Hypertrophy and Heart Failure

Laura Guilbert, Justine Dontaine, Natacha Fourny…·Luc Bertrand

This study identifies skNAC as critical for maintaining cardiomyocyte integrity and preventing heart failure by regulating proteome homeostasis and metabolic adaptation during pathological hypertrophy.

1 citation

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