Author

A. Herrlich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1

Frequent Co-Authors

E. Kefaloyianni· 4 shared A. Arthanarisami· 3 shared L. Ning· 3 shared D. Kreisel· 2 shared J. J. Atkinson· 2 shared M. M. Abdelmageed· 2 shared W. Li· 2 shared Y. Komaru· 2 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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4 shared papersPhysiology

E. Kefaloyianni

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4 papers

Recent·Most cited

Acute kidney injury triggers hypoxemia by inducing intravascular neutrophil retention that reduces lung capillary blood flow

Y. Komaru, L. Ning, C. Lama, A. Suresh, E. Kefaloyianni, M. J. Miller, S. Kawana, H. M. Shepherd, W. Li, D. Kreisel, A. Herrlich

This study identifies that acute kidney injury causes hypoxemia through intravascular neutrophil retention in lung capillaries, reducing blood flow without lung edema.

bioRxiv · Feb 27, 2024Physiology

Acute kidney injury-induced circulating TNFR1/2 elevations correlate with persistent kidney injury and progression to fibrosis

A. Arthanarisami, Y. Komaru, C. Katsouridi, J. Schumacher, D. K. Verges, L. Ning, M. M. Abdelmageed, A. Herrlich, E. Kefaloyianni

This study shows that acute kidney injury (AKI) increases circulating TNFR1/2 levels, which correlate with kidney injury severity and progression to fibrosis, particularly through extracellular vesicle-bound forms.

bioRxiv · Apr 3, 2023Physiology

Circulating osteopontin released by injured kidneys causes pulmonary inflammation and edema

F. Z. Khamissi, L. Ning, E. Kefaloyianni, H. Dun, A. Arthanarisami, A. Keller, J. J. Atkinson, W. Li, B. W. Wong, S. Dietmann, K. Lavine, D. Kreisel, A. Herrlich

This study identifies osteopontin (OPN) released by injured kidneys as a mediator of acute lung injury, showing its role in causing pulmonary inflammation and edema.

bioRxiv · Jul 20, 2021 · PreprintCell Biology

EGFR-driven profibrotic signals in AKI-to-CKD transition are blocked by TNF inhibition: Opportunities for etanercept treatment

M. M. Abdelmageed, E. Kefaloyianni, A. Arthanarisami, F. Khamissi, J. J. Atkinson, A. Herrlich

This study shows that TNF inhibition with etanercept prevents AKI-to-CKD transition and fibrosis as effectively as EGFR inhibition in mice, suggesting TNF as a downstream effector of EGFR-driven profibrotic signals.

bioRxiv · Apr 29, 2021 · PreprintCell Biology