Author

A. Javaheri

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pharmacology & Toxicology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Kovacs· 3 shared A. Diab· 2 shared A. Diwan· 2 shared A. Girardi· 2 shared C. Christoffersen· 2 shared C. Valenzuela Ripoll· 2 shared D. R. Rawnsley· 2 shared K. B. Margulies· 2 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersPhysiology

A. Kovacs

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Dapagliflozin improves endothelial integrity and hemodynamics in endotoxin treated mice through an apolipoprotein M dependent pathway

C. Valenzuela Ripoll, Z. Guo, T. Kumari, K. N. Miyata, M. Ozcan, A. Diab, A. Girardi, L. He, A. Kovacs, C. J. Weinheimer, J. Nigro, J. Oscarsson, R. Esterline, J. Schilling, M. Kosiborod, , A. Javaheri

This study shows dapagliflozin improves hemodynamics and endothelial integrity in endotoxin-treated mice via the ApoM/S1P pathway by preserving megalin expression.

bioRxiv · Apr 27, 2022 · PreprintPhysiology

TRAF2, an innate immune sensor, reciprocally regulates mitophagy and inflammation to maintain cardiac myocyte homeostasis.

X. Ma, D. R. Rawnsley, A. Kovacs, M. Islam, J. T. Murphy, C. Zhao, M. Kumari, L. M. Foroughi, H. Liu, K. Hyrc, S. Evans, B. A. French, K. B. Margulies, A. Javaheri, B. Razani, , A. Diwan

This study shows TRAF2 regulates mitophagy and inflammation in cardiac myocytes to prevent cell death and maintain homeostasis.

bioRxiv · Jun 12, 2021Cell Biology

Apolipoprotein M attenuates anthracycline cardiotoxicity and lysosomal injury

Z. Guo, C. Valenzuela Ripoll, A. Picataggi, D. R. Rawnsley, M. Ozcan, J. A. Chirinos, E. Chendamarai, A. Girardi, T. Riehl, H. Evie, A. Diab, A. Kovacs, K. Hyrc, X. Ma, A. Asnani, , A. Javaheri

This paper investigates the role of apolipoprotein M in anthracycline cardiotoxicity, finding that ApoM protects against cardiac dysfunction and lysosomal injury through the autophagy-lysosome pathway.

bioRxiv · Jan 12, 2021Pharmacology & Toxicology