Author

A. Koessinger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

D. Koessinger· 2 shared K. Stevenson· 2 shared A. Chalmers· 1 shared A. J. Chalmers· 1 shared C. Cloix· 1 shared C. Nixon· 1 shared D. Novo· 1 shared D. Zerbst· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

D. Koessinger

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Glioblastoma extracellular vesicles influence glial cell hyaluronic acid deposition to promote invasiveness

J. C. Norman, D. Koessinger, D. Novo, J. Birch, L. Mitchell, M. Moore, D. Zerbst, A. Chalmers, A. Koessinger, K. Stevenson, M. Nielson

This study identifies how GBM extracellular vesicles containing podocalyxin promote astrocyte hyaluronic acid deposition, enhancing GBM invasiveness.

bioRxiv · Feb 11, 2022 · PreprintCancer Biology

Increased apoptotic priming of glioblastoma enables therapeutic targeting by BH3-mimetics

A. Koessinger, D. Koessinger, K. Kinch, L. Martinez-Escardo, N. Paul, Y. Elmasry, G. Malviya, C. Cloix, K. Campbell, F. Bock, J. O'Prey, K. Stevenson, C. Nixon, M. Jackson, G. Ichim, , S. Tait

This study shows that glioblastoma (GBM) is highly sensitive to BH3-mimetics due to elevated anti-apoptotic BCL-xL and MCL-1 expression, which can be therapeutically targeted.

bioRxiv · Jun 13, 2021 · PreprintCancer Biology