Author

A. Rendek

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:flow cytometry· 2 Organism:human· 2 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

D. Ahern· 2 shared Z. Soonawalla· 2 shared A. Easton· 1 shared E. Abu-Shah· 1 shared E. H. Arbe-Barnes· 1 shared E. H. Ervin· 1 shared E. Kurz· 1 shared F. Liu· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

D. Ahern

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

High-dimensional mass cytometry reveals stemness state heterogeneity in pancreatic ductal adenocarcinoma

E. H. Ervin, D. Ahern, F. Liu, A. Rendek, Z. Soonawalla, U. Oppermann, P. Hulley, S. Pauklin

The paper uses mass cytometry to analyze stemness states in PDAC, revealing molecular heterogeneity and identifying a subset with plastic phenotypes.

bioRxiv · Feb 27, 2024 · PreprintCancer Biology

Immune responses in pancreatic cancer may be restricted byprevalence of activated regulatory T-cells, dysfunctionalCD8+ T-cells, and senescent T-cells

S. Sivakumar, E. Abu-Shah, D. Ahern, E. H. Arbe-Barnes, N. Mangal, S. Reddy, A. Rendek, A. Easton, E. Kurz, M. Silva, L. R. Heij, Z. Soonawalla, R. Bashford-Rogers, M. R. Middleton, M. Dustin

This study identifies immune cell states in pancreatic cancer, showing regulatory T-cells and dysfunctional CD8+ T-cells contribute to a suppressive microenvironment, suggesting new therapeutic targets.

bioRxiv · Jun 20, 2020 · PreprintImmunology