Author

A. Thakurta

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Organism:human· 3 Research Focus:gene regulatory network inference· 2 Approach:benchmark study· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Approach:deep learning· 1

Frequent Co-Authors

B. A. Walker· 2 shared D. Auclair· 2 shared E. Flynt· 2 shared F. Towfic· 2 shared G. J. Morgan· 2 shared H. Avet-Loiseau· 2 shared K. Mavrommatis· 2 shared M. Samur· 2 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

B. A. Walker

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Integrative multi-omics identifies high risk Multiple Myeloma segment associated with significant DNA loss and dysregulated DNA repair and cell cycle pathways

M. Ortiz Estevez, M. Samur, F. Towfic, E. Flynt, N. Stong, I. S. Jang, K. Wang, P. Vyas, N. Munshi, H. Avet-Loiseau, M. W. Trotter, G. J. Morgan, B. Walker, A. Thakurta

The paper identifies a high-risk Multiple Myeloma subgroup using multi-omics data, linking genomic loss to dysregulated DNA repair and cell cycle pathways.

bioRxiv · Sep 3, 2021Cancer Biology

Multiple Myeloma DREAM Challenge Reveals Epigenetic Regulator PHF19 As Marker of Aggressive Disease

M. J. Mason, C. Schinke, C. Eng, F. Towfic, F. Gruber, A. Dervan, B. S. White, A. Pratapa, Y. Guan, H. Chen, Y. Cui, B. Li, T. Yu, E. Chaibub Neto, K. Mavrommatis, , J. A. Guinney

The paper identifies PHF19 as a marker of aggressive multiple myeloma and shows a simple four-feature model performs as well as complex gene expression models.

bioRxiv · Aug 22, 2019Cancer Biology

Microhomology-mediated end joining drives complex rearrangements and over expression of MYC and PVT1 in multiple myeloma

A. Mikulasova, C. Ashby, R. G. Tytarenko, P. Qu, A. Rosenthal, J. A. Dent, K. R. Ryan, M. A. Bauer, C. P. Wardell, A. Hoering, K. Mavrommatis, M. Trotter, S. Deshpande, S. Yaccoby, E. Tian, , B. A. Walker

The paper identifies MYC rearrangements in multiple myeloma, showing they are complex and linked to overexpression of MYC and PVT1 through distinct DNA repair mechanisms.

bioRxiv · Jun 24, 2019Cancer Biology