Author

A. Z. L. Shih

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. Chaudhry· 1 shared B. Hoffman· 1 shared B. Vanderkruk· 1 shared C. B. Verchere· 1 shared D. J. Pasula· 1 shared D. S. Luciani· 1 shared R. Shi· 1 shared T. Willnow· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. Chaudhry

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2 papers

Recent·Most cited

Cell BiologybioRxivApr 21, 2022

SORLA mediates endocytic uptake of proIAPP and protects against islet amyloid deposition

A. Z. L. Shih, Y.-C. Chen, C. B. Verchere, T. Willnow

This study shows SORLA reduces islet amyloid deposition by mediating endocytic uptake of proIAPP, preventing cell death in pancreatic beta cells.

DOI 10.1016/j.molmet.2022.101585

mouseproteomics

𝕏 PostBluesky

Cell BiologybioRxivPreprintOct 25, 2021

Bcl-xL restricts transcriptional, morphological and functional decompensation of β-cell mitochondria under chronic glucose excess

D. J. Pasula, R. Shi, B. Vanderkruk…·D. S. Luciani

This study shows that Bcl-xL prevents mitochondrial dysfunction in beta-cells under chronic glucose stress by limiting mitochondrial ROS and maintaining gene expression.

DOI 10.1101/2021.10.25.465491

mousegenetic perturbation / CRISPR

𝕏 PostBluesky