Author

B. B. Kahn

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:metabolism & bioenergetics· 2 Assay:metabolomics· 1 Assay:metagenomics· 1

Frequent Co-Authors

A. Banks· 1 shared A. Castoldi· 1 shared A. I. Mina· 1 shared A. M. Pinto· 1 shared A. Rahnavard· 1 shared A. Saghatelian· 1 shared A. Santoro· 1 shared A. T. Nelson· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperCell Biology

A. Banks

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Beneficial metabolic effects of PAHSAs depend on the gut microbiota in diet-induced obese mice

J. Lee, K. Wellenstein, A. Rahnavard, A. T. Nelson, M. Holter, B. P. Cummings, V. Yeliseyev, A. Castoldi, C. B. Clish, L. Bry, D. Siegel, B. B. Kahn

This study shows that PAHSAs improve insulin sensitivity in diet-induced obese mice via the gut microbiota, with sex-specific effects observed.

bioRxiv · Sep 28, 2023 · PreprintPhysiology

Inflammation causes insulin resistance via interferon regulatory factor 3 (IRF3)-mediated reduction in FAHFA levels

S. Yan, A. Santoro, M. J. Niphakis, A. M. Pinto, C. L. Jacobs, R. Ahmad, R. M. Suciu, B. R. Fonslow, R. B. Herbst-Graham, N. Ngo, C. L. Henry, D. M. Herbst, A. Saghatelian, B. B. Kahn, E. D. Rosen

This study identifies the IRF3/AIG1 axis in adipocytes as a mechanism linking obesity-induced inflammation to insulin resistance and suggests AIG1 inhibition as a therapeutic approach.

bioRxiv · Aug 8, 2023 · PreprintPhysiology

Obesity-linked PPARγ S273 phosphorylation promotes insulin resistance through Growth Differentiation Factor 3

J. A. Hall, D. Ramachandran, H. C. Roh, J. R. DiSpirito, T. Belchior de Oliveira, P.-J. H. Zushin, C. J. Palmer, S. Hong, A. I. Mina, B. Liu, Z. Deng, P. Aryal, C. Jacobs, D. Tenen, C. W. Brown, , A. Banks

This study shows that preventing PPARγ S273 phosphorylation reduces insulin resistance without weight gain or side effects, identifying Gdf3 as a key mediator.

bioRxiv · Jan 13, 2020Cell Biology