Author

B. Lapin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

F. Bienaime· 2 shared M. Mazloum· 2 shared S. Coscoy· 2 shared S. Descroix· 2 shared A. Aka· 1 shared A. P. Glaser· 1 shared A. Viau· 1 shared B. Lelongt· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersBiophysics

F. Bienaime

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Author profile →

3 papers

Recent·Most cited

Decoupling shear stress and pressure effects in the biomechanics of autosomal dominant polycystic kidney disease using a perfused kidney-on-chip.

B. Lapin, G. Gropplero, J. Vandensteen, M. Mazloum, F. Bienaime, S. Descroix, S. Coscoy

This study uses a kidney-on-chip model to decouple shear stress and pressure effects on tubule dilation in Pkd1-/- cells, revealing distinct mechanisms in different kidney cell types.

bioRxiv · Jun 18, 2024Biophysics

Cilia to basement membrane signalling is a biomechanical driver of autosomal dominant polycystic kidney disease

M. Mazloum, B. Lapin, A. Viau, R. Alghamdi, M. Burtin, P. Houillier, L. Cheval, G. Crambert, A. Aka, E. W. Kuehn, C. Cohen, S. Descroix, T. Busch, M. Koettgen, S. Garbay, , F. Bienaime

This study identifies biomechanical changes in the tubular basement membrane as drivers of cyst formation in ADPKD, linking cilia and polycystin-1 function.

bioRxiv · Jun 6, 2024 · PreprintPhysiology

TNF Blockade Reduces Prostatic Hyperplasia and Inflammation while Limiting BPH Diagnosis in Patients with Autoimmune Disease

R. E. Vickman, L. Aaron-Brooks, R. Zhang, N. A. Lanman, B. Lapin, V. Gil, M. Greenberg, T. Sasaki, G. M. Cresswell, M. M. Broman, J. Petkewicz, P. Talaty, B. T. Helfand, A. P. Glaser, C.-H. Wang, , S. W. Hayward

Patient data and mouse models suggest that repurposing tumor necrosis factor alpha blockade reduces inflammation-mediated prostatic hyperplasia.

bioRxiv · Mar 11, 2021Cell Biology