Author

D. Nakano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Organism:rat· 1

Frequent Co-Authors

A. Nishiyama· 3 shared A. Kanai· 1 shared A. Saito· 1 shared H. Hanaoka· 1 shared H. Ikeuchi· 1 shared H. Ishii· 1 shared H. Matsui· 1 shared H. Obinata· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersBiochemistry

A. Nishiyama

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Author profile →

3 papers

Recent·Most cited

Robust capability of renal tubule fatty acid uptake from apical and basolateral membranes in physiology and disease

R. Kawakami, H. Hanaoka, A. Kanai, H. Obinata, D. Nakano, H. Ikeuchi, M. Matsui, T. Matsuzaki, R. Tanaka, H. Sunaga, S. Goto, H. Matsui, N. Koitabashi, K. Saegusa, T. Yokoyama, , T. Iso

This study investigates fatty acid uptake by renal tubule epithelial cells in mice, showing uptake from both blood and urine, and storage as neutral lipids, with CD36 playing a key role in blood-derived uptake.

bioRxiv · Jul 4, 2022 · PreprintBiochemistry

Mass Recovery following Caloric Restriction Reverses Lipolysis and Proteolysis, but not Gluconeogenesis, in Insulin Resistant OLETF Rats

M. A. Cornejo, J. Dhillon, A. Nishiyama, D. Nakano, R. M. Ortiz

This study examines how caloric restriction and subsequent mass recovery affect substrate metabolism in insulin-resistant rats, finding that lipolysis and proteolysis are reversed, but gluconeogenesis remains unchanged.

bioRxiv · May 17, 2021Physiology

The p21 dependent G2 arrest of the cell cycle in epithelial tubular cells links to the early stage of renal fibrosis

T. Koyano, M. Namba, T. Kobayashi, K. Nakakuni, D. Nakano, M. Fukushima, A. Nishiyama, M. Matsuyama

The paper identifies p21-dependent G2 cell cycle arrest in proximal tubular cells as an early response to renal damage, which may protect kidney function during the initial stage of fibrosis.

bioRxiv · Feb 26, 2019Cell Biology