Author

E. Mohr

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1 Pathology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Organism:rat· 2 Organism:chicken / avian· 1 Organism:human· 1

Frequent Co-Authors

A. Weiler· 2 shared E. Peterson· 2 shared G. Barry· 2 shared J. Osorio· 2 shared M. Semler· 2 shared N. Schultz-Darken· 2 shared S. Capuano· 2 shared T. Friedrich· 2 shared

Activity by Year

2017-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersPathology

A. Weiler

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

SGLT2 inhibitors attenuate endothelial to mesenchymal transition and cardiac fibroblast activation

K. Schmidt, A. Schmidt, S. Gross, A. Just, A. Pfanne, M. Fuchs, M. Jordan, E. Mohr, A. Pich, J. Fiedler, T. Thum

This study shows SGLT2 inhibitors reduce endothelial to mesenchymal transition and cardiac fibroblast activation through effects on mitochondrial respiration, actin cytoskeleton, and specific transporters.

bioRxiv · Dec 18, 2023Pharmacology & Toxicology

Stromal cell-contact dependent PI3K and APRIL induced NF-κB signaling complement each other to prevent mitochondrial- and endoplasmic reticulum stress induced cell death of bone marrow plasma cells

R. Cornelis, S. Hahne, A. Taddeo, G. Petkau, D. Malko, P. Durek, M. Thiem, L. Heiberger, E. Mohr, C. Klaeden, K. Tokoyoda, F. Siracusa, B. F. Hoyer, F. Hiepe, M.-F. Mashreghi, , A. Radbruch

The paper shows that stromal cell contact and APRIL signaling via PI3K and NF-κB pathways prevent mitochondrial and ER stress-induced apoptosis in bone marrow plasma cells.

bioRxiv · Nov 21, 2019Immunology

Primary infection with dengue or Zika virus does not affect the severity of heterologous secondary infection in macaques.

M. E. Breitbach, C. M. Newman, D. M. Dudley, L. M. Stewart, M. T. Aliota, M. R. Koenig, P. Shepherd, K. Yamamoto, C. Crooks, G. Young, M. Semler, A. Weiler, G. Barry, H. Heimsath, E. Mohr, , D. H. O'Connor

This study investigates whether prior dengue or Zika virus infection affects the severity of subsequent heterologous secondary infections in macaques, finding no significant impact on clinical outcomes.

bioRxiv · Apr 18, 2019Pathology

Infection via mosquito bite alters Zika virus replication kinetics in rhesus macaques

D. Dudley, C. Newman, J. Lalli, L. Stewart, M. Koening, A. Weiler, M. Semler, G. Barry, K. Zarbock, M. Mohns, M. Breitbach, N. Schultz-Darken, E. Peterson, W. Newton, E. Mohr, , M. Aliota

This study compares ZIKV replication kinetics and tissue distribution in rhesus macaques infected via mosquito bite versus subcutaneous inoculation, finding delayed viral peak and restricted tissue distribution in mosquito-infected animals.

bioRxiv · Sep 8, 2017Microbiology