Author

E. S. Halstead

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Bonavia· 1 shared A. C. Arnold· 1 shared C. Dong· 1 shared E. Yau· 1 shared G. Li· 1 shared H. M. Atkins· 1 shared K. Fino· 1 shared K. Hassan· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperBioengineering

A. Bonavia

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2 papers

Recent·Most cited

Mesenchymal stromal cells and alpha-1 antitrypsin have a strong synergy in modulating inflammation and its resolution

L. Han, X. Wu, O. Wang, X. Luan, W. H. Velander, M. Aynardi, E. S. Halstead, A. Bonavia, R. Jin, G. Li, Y. Li, Y. Wang, C. Dong, Y. Lei

This study shows that combining mesenchymal stromal cells and alpha-1 antitrypsin significantly improves inflammation modulation and resolution in vitro and in a mouse model.

bioRxiv · Nov 19, 2022 · PreprintBioengineering

Disruption of the surfactant protein A receptor SP-R210L (CD245α/MYO18Aα) alters respiratory function and iron sequestration in alveolar macrophages of aged mice

E. Yau, T. M. Umstead, R. Abdulqadir, K. Fino, Z. Guan, S. Hu, S. Diangelo, K. Hassan, S. Bingaman, H. M. Atkins, T. K. Cooper, A. C. Arnold, E. S. Halstead, Z. C. Chroneos

This study shows that disrupting the SP-R210L isoform in aged mice impairs respiratory function and iron sequestration in alveolar macrophages, leading to chronic dysregulation and increased interstitial lung disease risk.

bioRxiv · Apr 14, 2021 · PreprintPhysiology