Author

H. M. Silva

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

D. Li· 2 shared D. R. Littman· 2 shared L. Kroehling· 2 shared L. Wu· 2 shared W.-y. Lin· 2 shared A. C. Kimmelman· 1 shared A. Hine· 1 shared C. Au· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersImmunology

D. Li

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2 papers

Recent·Most cited

ImmunologybioRxivJun 10, 2020

Niche-selective inhibition of pathogenic Th17 cells by targeting metabolic redundancy

L. Wu, K. E. R. Hollinshead, Y. Hao…·D. R. Littman

This study shows that targeting Gpi1 selectively inhibits pathogenic Th17 cells in autoimmune diseases while sparing homeostatic Th17 cells.

DOI 10.1016/j.cell.2020.06.014

mousebulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivJun 25, 2019

Serum Amyloid A Proteins Induce Pathogenic TH17 Cells and Promote Inflammatory Disease

J.-Y. Lee, J. A. Hall, L. Kroehling…·D. R. Littman

This study shows serum amyloid A proteins drive pathogenic TH17 cell differentiation and promote inflammatory disease through STAT3-activating cytokines.

DOI 10.1016/j.cell.2019.11.026

mouseexperimental assay

𝕏 PostBluesky