Author

J.-M. Fustin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Evolutionary Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Hayes· 1 shared B. Ananthasubramaniam· 1 shared B. Saer· 1 shared C. H. Johnson· 1 shared C. Rakers· 1 shared D. A. Golombek· 1 shared D. Whitmore· 1 shared G. Taylor· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPhysiology

A. Hayes

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Dietary methionine/choline deficiency affects behavioural and molecular circadian rhythms

B. Saer, G. Taylor, A. Hayes, B. Ananthasubramaniam, J.-M. Fustin

This study shows that dietary methionine/choline deficiency disrupts circadian rhythms and gene expression in mice, with effects dependent on circadian time.

bioRxiv · Jan 4, 2024 · PreprintPhysiology

Hyperpolyploidization of hepatocyte initiates preneoplastic lesion formation in the liver

H.-W. Chao, H. Lin, Y.-S. Huang, J.-M. Fustin, M. Doi, H. Chen, H.-H. Lai, S.-H. Lin, Y.-L. Lee, P.-C. King, H.-S. Hou, H.-W. Chen, P.-Y. Young

This study shows that hyperpolyploidization of hepatocytes in the centrilobular region initiates preneoplastic lesion formation, with AURKB upregulation and phosphorylation driving this process in HCC development.

bioRxiv · Aug 23, 2020Pathology

The methyl cycle is a conserved regulator of biological clocks.

J.-M. Fustin, S. Ye, C. Rakers, M. Versteven, S. J. Cargill, K. T. Tamai, Y. Xu, M. L. Jabbur, R. Kojima, M. L. Lamberti, D. Whitmore, R. Stanewsky, R. Kageyama, T. Matsuo, K. Yoshioka-Kobayashi, , H. Okamura

This study shows the methyl cycle regulates circadian and somite segmentation clocks across species, highlighting its role as a conserved metabolic regulator of biological clocks.

bioRxiv · May 29, 2019 · PreprintEvolutionary Biology