Author

M.-s. Lee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Bioengineering· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Adler· 1 shared A. Sivapatham· 1 shared B. W. Nemke· 1 shared C. A. Lewis· 1 shared C. B. Clish· 1 shared D. Brooks· 1 shared D. S. Hitchcock· 1 shared E. M. Leiferman· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Adler

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

ER-to-lysosome Ca2+ refilling followed by K+ efflux-coupled store-operated Ca2+ entry in inflammasome activation and metabolic inflammation

H. Kang, S. W. Choi, J. Y. Kim, S.-J. Oh, S. J. Kim, M.-s. Lee

This study identifies a novel mechanism involving lysosomal Ca2+ release and K+ efflux through KCa3.1 channels in inflammasome activation and metabolic inflammation.

eLife · Jul 2, 2024Peer-reviewedImmunology

Autologous iPSC- and MSC-derived Chondrocyte Implants for Cartilage Repair in a Miniature Pig Model

M.-S. Lee, A. Sivapatham, E. M. Leiferman, H. Jiao, Y. Lu, B. W. Nemke, M. Leiferman, M. D. Markel, W.-J. Li

This study compares autologous iPSC- and BMSC-derived chondrocyte implants in minipigs, finding iMSC-Ch implants produce more hyaline cartilage-like tissue.

bioRxiv · Jul 25, 2023Bioengineering

CRTC2 regulates plasma cell metabolism and survival

J. Hong, F. Ahsan, E. Montecino-Rodriguez, P. Pioli, M.-s. Lee, T. Nguyen, D. Brooks, J. Golovato, K. Niazi, K. Dorshkind, M. Teitell

This study shows that CRTC2 repression is essential for plasma cell metabolism and survival, affecting antibody production and immune response durability.

bioRxiv · Apr 14, 2021 · PreprintImmunology

Adaptation of pancreatic cancer cells to nutrient deprivation is reversible and requires glutamine synthetase stabilization by mTORC1

P.-Y. Tsai, M.-S. Lee, U. Jadhav, I. Naqvi, S. Madha, A. Adler, M. Mistry, S. Naumenko, C. A. Lewis, D. S. Hitchcock, F. R. Roberts, P. DelNero, T. Hank, K. C. Honselmann, V. Morales Oyarvide, , N. Y. Kalaany

This study shows that pancreatic cancer cells can adapt to nutrient deprivation by stabilizing glutamine synthetase via mTORC1, and this adaptation is reversible.

bioRxiv · Feb 16, 2020Cancer Biology