Author

M. Pour

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

I. Yanai· 3 shared A. Rao· 2 shared B. Xia· 2 shared D. Barkley· 2 shared G. S. Franca· 2 shared M. Baron· 2 shared A. Lund· 1 shared A. Miller· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersCancer Biology

I. Yanai

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Drug-induced adaptation along a resistance continuum in cancer cells

G. S. Franca, M. Baron, M. Pour, B. R. King, A. Rao, S. Misirlioglu, D. Barkley, I. Dolgalev, K. Ho-Tang, G. Avital, F. Kuperwaser, A. Patel, D. A. Levine, T. Lionnet, I. Yanai

This study shows that gradual dose exposure and prolonged treatment drive ovarian cancer cell resistance to targeted therapy through a continuous adaptation process involving transcriptional, epigenetic, and genetic changes.

bioRxiv · Jun 21, 2022Cancer Biology

Recurrence of cancer cell states across diverse tumors and their interactions with the microenvironment

D. Barkley, R. Moncada, M. Pour, D. Liberman, I. Dryg, G. Werba, W. Wang, M. Baron, A. Rao, B. Xia, G. S. Franca, A. Weil, D. F. Delair, C. Hajdu, A. Lund, , I. Yanai

The paper identifies recurrent cancer cell states across 15 tumor types and examines their interactions with the tumor microenvironment using single-cell RNA-Seq and spatial transcriptomics.

bioRxiv · Dec 20, 2021 · PreprintCancer Biology

The genetic basis of tail-loss evolution in humans and apes

B. Xia, W. Zhang, A. Wudzinska, E. Huang, R. Brosh, M. Pour, A. Miller, J. S. Dasen, M. T. Maurano, S. Y. Kim, J. D. Boeke, I. Yanai

This paper identifies an Alu element insertion in the TBXT gene as the genetic mechanism driving tail-loss evolution in hominoids, showing it causes alternative splicing and tail absence in mice.

bioRxiv · Sep 14, 2021Evolutionary Biology