Author

P. Castets

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

M. CASTETS· 2 shared M. Creveaux· 2 shared M. Theodore· 2 shared P. Huchede· 2 shared A. Bertrand-Chapel· 1 shared A. Bouvier· 1 shared A. Dutour· 1 shared A. E. M. Kneppers· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCancer Biology

M. CASTETS

Mutual co-authors in cluster

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4 papers

Recent·Most cited

mTORC1-dependent SOCE activity regulates synaptic gene expression and muscle response to denervation

A. Prola, O. Dupont, J. Rajendran, F. Chabry, S. Koenig, M. Frieden, P. Castets

The paper identifies SOCE's role in synaptic gene regulation and muscle response to denervation, showing mTORC1's involvement in this process.

bioRxiv · Apr 1, 2024 · PreprintPhysiology

Destabilization of neuromuscular junctions and deregulation of activity-dependent signalling pathways in Myotonic Dystrophy type I

D. Falcetta, S. Quirim, I. Cocchiararo, M. Cornut, M. Theodore, A. Stiefvater, S. Lin, L. Tintignac, R. Ivanek, J. Kinter, M. A. Ruegg, M. Sinnreich, P. Castets

This study investigates NMJ destabilization and activity-dependent signaling deregulation in DM1 mouse models, identifying CaMKII and HDAC4 as key factors in synaptic gene expression and muscle dysfunction.

bioRxiv · Mar 8, 2023 · PreprintPhysiology

Alterations of the TGFb-sequestration complex member ADAMTSL1 levels are associated with muscular defects and rhabdomyosarcoma aggressiveness

A. Bertrand-Chapel, S. Meyer, G. Juban, A. E. M. Kneppers, P. Huchede, C. Gallerne, R. Benayoun, E. Cohen, A. Lopez-Gonzalez, S. B. Larbi, M. Creveaux, L. Vaille, A. Bouvier, M. Theodore, L. Broutier, , M. CASTETS

This study shows ADAMTSL1 is involved in skeletal muscle regeneration and acts as a prognostic factor in aggressive rhabdomyosarcoma, correlating with a neural-like phenotype.

bioRxiv · Mar 7, 2023 · PreprintCancer Biology

Low expression of ANT1 confers oncogenic properties to rhabdomyosarcoma tumor cells via modulating metabolism and death pathways.

J. Vial, P. Huchede, S. Fagault, F. Basset, M. Rossi, J. Geoffray, J. Bisaccia, M. Creveaux, D. Neves, F. Fauvelle, P. Castets, M. Carre, K. Weber, M. Castets

This study shows that low ANT1 expression in rhabdomyosarcoma cells promotes oncogenic properties by altering metabolism and death pathways, and restoring ANT1 sensitizes cells to chemotherapy.

bioRxiv · Mar 3, 2020Cancer Biology