Author

S. Bannwarth

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

V. Paquis-Flucklinger· 3 shared E. C. Genin· 2 shared S. Lacas-Gervais· 2 shared A. Lefevre· 1 shared A. Mauri-Crouzet· 1 shared A. Petit-Paitel· 1 shared A. Singhi· 1 shared A.-S. Gay· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersCell Biology

V. Paquis-Flucklinger

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Multiomics study of CHCHD10S59L-related disease reveals energy metabolism downregulation: OXPHOS and beta-oxidation deficiencies associated with lipids alterations

B. Madji Hounoum, R. Bellon, E. C. Genin, S. Bannwarth, A. Lefevre, L. Fleuriol, D. Debayle, A.-S. Gay, A. Petit-Paitel, S. Lacas-Gervais, H. Blasco, P. Edmond, V. Paquis-Flucklinger, J.-E. RICCI

This study identifies metabolic and lipid alterations in CHCHD10S59L-related disease, showing OXPHOS and beta-oxidation deficiencies and suggesting L-acetylcarnitine as a potential therapeutic.

bioRxiv · Jan 19, 2023 · PreprintCell Biology

SLP2/prohibitins aggregates and instability of the PHB complex are key elements in CHCHD10S59L-related disease

E. C. GENIN, S. Bannwarth, B. Ropert, A. Mauri-Crouzet, F. Lespinasse, G. Auge, K. Fragaki, C. Cochaud, S. Lacas-Gervais, V. Paquis-Flucklinger

The paper identifies SLP2/prohibitin aggregates and PHB complex instability as key factors in CHCHD10S59L-related disease, leading to mitochondrial dysfunction and apoptosis.

bioRxiv · May 31, 2021 · PreprintPathology

Petite Integration Factor 1 (PIF1) helicase deficiency increases weight gain in Western diet-fed female mice without increased inflammatory markers or decreased glucose clearance

B. A. Kaufman, F. Belmonte, N. Dedousis, I. Sipula, N. Desai, A. Singhi, y. chu, Y. Zhang, S. Bannwarth, V. Paquis-Flucklinger, L. Harrington, R. M. O'Doherty, M. J. Jurczak

This study shows that PIF1 helicase deficiency in female mice on a Western diet leads to increased weight gain but not inflammation or reduced glucose clearance.

bioRxiv · Aug 16, 2018Physiology