Author

S. K. Plevritis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. J. Giaccia· 2 shared G. Bouchard· 2 shared J. B. Shrager· 2 shared W. Zhang· 2 shared A. Bermudez· 1 shared A. Bhattacharya· 1 shared A. Ribas· 1 shared A. Xu· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBioengineering

A. J. Giaccia

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Author profile →

3 papers

Recent·Most cited

BioengineeringbioRxivPreprintSep 11, 2023

The colocatome as a spatial -omic reveals shared microenvironment features between tumour-stroma assembloids and lung cancer specimens

G. Bouchard, W. Zhang, I. Li…·S. K. Plevritis

The paper introduces the colocatome, a spatial-omic framework, to compare microenvironment features between tumour-stroma assembloids and lung cancer specimens, finding that fibroblasts protect cancer cells from treatment by reorganizing cytoarchitecture.

DOI 10.1101/2023.09.11.557278

humanimaging

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Cancer BiologybioRxivPreprintMay 28, 2021

Multi-Omics Analysis of Fibroblasts from the Invasive Tumor Edge Reveals that Tumor-Stroma Crosstalk Induces O-glycosylation of the CDK4-pRB Axis

G. Bouchard, F. J. G. Marques, L. G. Karacosta…·S. K. Plevritis

The paper analyzes multi-omics data from fibroblasts at the tumor edge and core to show O-glycosylation of the CDK4-pRB axis is induced by tumor-stroma crosstalk.

DOI 10.1101/2021.05.28.446229

humanproteomics

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BioengineeringbioRxivPreprintSep 12, 2019

Trajectories from Snapshots: Integrated proteomic and metabolic single-cell assays reveal multiple independent adaptive responses to drug tolerance in a BRAF-mutant melanoma cell line

Y. Su, G. Li, M. E. Ko…·J. R. Heath

The paper uses integrated proteomic and metabolic single-cell assays to identify two distinct adaptive pathways in BRAF-mutant melanoma cells during drug treatment.

DOI 10.1101/767988

humanproteomics

𝕏 PostBluesky