Author

S. Miki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Assay:bulk RNA-seq· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Assay:clinical cohort / trial· 1 Research Focus:clustering· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

F. B. Furnari· 3 shared T. Koga· 3 shared A. D. Parisian· 2 shared P. S. Mischel· 2 shared A. D. Boyer· 1 shared A. Khan· 1 shared A. M. Armando· 1 shared A. Rzhetsky· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersCancer Biology

F. B. Furnari

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4 papers

Recent·Most cited

Glioblastoma Mesenchymal Transition and Invasion are Dependent on a NF-κB/BRD2 Chromatin Complex

r. vadla, S. Miki, B. Taylor, D. Kawauchi, B. M. Jones, N. Nathwani, P. Pham, J. Tsang, D. A. Nathanson, F. B. Furnari

This study identifies the PTEN-NF-κB-BRD2 pathway as critical for glioblastoma mesenchymal transition and invasion, suggesting BET-BD2 inhibitors as a therapeutic strategy.

bioRxiv · Jul 3, 2023 · PreprintCancer Biology

Targeting Glioblastoma Signaling and Metabolism with A Re-Purposed Brain-Penetrant Drug

J. Bi, A. Khan, J. Tang, S. Wu, W. Zhang, R. C. Gimple, T. Koga, A. M. Armando, S. Miki, H. Yang, B. Prager, E. J. Curtis, D. A. Wainwright, F. B. Furnari, J. N. Rich, , P. S. Mischel

This study identifies fluoxetine as a potential therapy for glioblastoma by inhibiting SMPD1 and enhancing temozolomide efficacy, with real-world evidence suggesting improved survival.

bioRxiv · Mar 16, 2021Cancer Biology

SMARCB1 loss interacts with neuronal differentiation state to block maturation and impact cell stability

A. D. Parisian, T. Koga, S. Miki, P. D. Johann, M. Kool, J. R. Crawford, F. Furnari

This study shows SMARCB1 loss affects neuronal differentiation and cell stability differently based on the differentiation state, contributing to ATRT tumorigenesis.

bioRxiv · May 4, 2020Cancer Biology

Cancer avatars derived from genetically engineered pluripotent stem cells allow for longitudinal assessment of tumor development

T. Koga, J. A. Benitez, I. A. Chaim, S. Markmiller, A. D. Parisian, R. F. Hevner, K. M. Turner, F. M. Hessenauer, M. D'Antonio, N.-p. D. Nguyen, S. Saberi, J. Ma, S. Miki, A. D. Boyer, J. Ravits, , F. B. Furnari

This paper introduces cancer avatar models using genetically engineered hiPSCs to study tumor development and heterogeneity in glioblastoma.

bioRxiv · Oct 25, 2019Cancer Biology