Author

S. Oki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 3 Research Focus:chromatin & epigenetic regulation· 2 Approach:deep learning· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. E. Frazier· 1 shared A. Hayashi· 1 shared A. Otake· 1 shared A. P. Makohon-Moore· 1 shared A. Takeda· 1 shared A. Zucker· 1 shared C. A. Iacobuzio-Donahue· 1 shared D. R. Thorburn· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

A. E. Frazier

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Atf3 controls transitioning in female mitochondrial cardiomyopathy as identified by single-cell transcriptomics

T. Qaqorh, Y. Takahashi, K. Otani, I. Yazawa, Y. Nishida, Y. Fujihara, M. Honda, S. Oki, Y. Ohkawa, D. R. Thorburn, A. E. Frazier, A. Takeda, Y. Ikeda, H. Sakaguchi, T. Watanabe, , Y. Shintani

This study identifies Atf3 as a key regulator of transition from compensation to dysfunction in female mitochondrial cardiomyopathy using single-cell transcriptomics.

bioRxiv · Sep 11, 2023Cell Biology

Elucidating disease-associated mechanisms triggered by pollutants via the epigenetic landscape using large-scale ChIP-Seq data

Z. Zou, Y. Yoshimura, Y. Yamanishi, S. Oki

The paper introduces DAR-ChIPEA, a method using ChIP-Seq data to identify TFs involved in pollutant-induced epigenetic changes, outperforming previous methods in predicting disease mechanisms.

bioRxiv · May 18, 2023Pharmacology & Toxicology

Evolutionary Dynamics of Non-Coding Regions in Pancreatic Ductal Adenocarcinoma

A. Hayashi, Y.-j. Ho, A. P. Makohon-Moore, A. Zucker, J. Hong, J. G. Reiter, J. Huang, L. Zhang, M. A. Attiyeh, P. Baez, R. Kappagantula, J. P. Melchor, E. M. OReilly, N. D. Socci, S. Oki, , C. A. Iacobuzio-Donahue

This study investigates the evolutionary dynamics of non-coding regions in pancreatic cancer, finding higher mutational burden in noncoding DNA and similar mutational spectra between enhancers and coding DNA.

bioRxiv · Sep 11, 2020 · PreprintCancer Biology