Author

T. Sulpice

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1

Frequent Co-Authors

F. Briand· 2 shared A. Deney· 1 shared A. J. Finch· 1 shared A. Montagner· 1 shared A. Tailleux· 1 shared B. Emerson· 1 shared B. Staels· 1 shared B. Tramunt· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

F. Briand

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Crest maturation at the cardiomyocyte surface contributes to a new late postnatal development stage that controls the diastolic function of the adult heart

c. karsenty, c. frugier, G. GENET, M.-H. SEGUELAS, P. Alzieu, O. Cazorla, A. Montagner, Y. Blum, c. Dubroca, J. Maupoint, B. Tramunt, M. Cauquil, T. Sulpice, S. Richard, S. Arcucci, , C. Gales

This study identifies a late postnatal developmental stage in rodents where cardiomyocyte surface crests mature, regulating diastolic function through ephrin-B1-dependent mechanisms.

bioRxiv · Feb 11, 2022Cell Biology

The hepatic compensatory response to elevated systemic sulfide promotes diabetes

R. N. Carter, M. T. G. Gibbins, M. E. Barrios-Llerena, S. E. Wilkie, P. L. Freddolino, M. Libiad, V. Vitvitsky, B. Emerson, T. Le Bihan, M. Brice, H. Su, S. G. Denham, N. Z. M. Homer, C. Mc Fadden, A. Tailleux, , N. M. Morton

This study shows that mice lacking the sulfide oxidation pathway enzyme TST exhibit metabolic abnormalities despite insulin sensitization, highlighting TST's role in hepatic metabolism and potential implications for sulfide-donor therapies.

bioRxiv · Apr 27, 2020Physiology

Liraglutide improves hepatic steatosis and metabolic dysfunctions in a 3-week dietary mouse model of non-alcoholic steatohepatitis

T. Duparc, F. Briand, C. Trenteseaux, J. Merian, G. Combes, S. Najib, T. Sulpice, L. O. Martinez

This study develops a 3-week dietary mouse model of NASH and shows liraglutide reduces hepatic steatosis and inflammation.

bioRxiv · May 17, 2019Pathology