Author

V. Brusic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:cryo-EM· 1 Organism:human· 1 Organism:multiple species· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. Coscia· 1 shared C. J. Mann· 1 shared E. Cassella· 1 shared F. Schmidt· 1 shared G. Tekes· 1 shared H. Varnum· 1 shared J. Abraham· 1 shared J. L. Timpona· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperMicrobiology

A. Coscia

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Structural basis for VLDLR recognition by eastern equine encephalitis virus

P. Yang, W. Li, X. Fan, J. Pan, C. J. Mann, H. Varnum, L. E. Clark, S. A. Clark, A. Coscia, K. N. Smith, V. Brusic, J. Abraham

This study determines the structures of EEEV and SFV spike glycoproteins bound to VLDLR, revealing distinct binding modes that explain how different alphaviruses use the same receptor.

bioRxiv · Nov 14, 2023Microbiology

The ancient fusogen EnvP(b)1 is expressed in human tissues and its structure informs the evolution of gammaretrovirus envelope proteins

K. R. McCarthy, J. L. Timpona, S. Jenni, V. Brusic, W. E. Johnson, S. P. J. WHELAN, L. R. Robinson-McCarthy

The paper identifies EnvP(b)1, an ancient retroviral envelope gene expressed in human tissues, and shows its structural similarity to modern leukemia virus Envs, suggesting a conserved scaffold for receptor binding evolution.

bioRxiv · Apr 22, 2020 · PreprintMicrobiology

Vesicular stomatitis virus transcription is inhibited by TRIM69 in the interferon-induced antiviral state

T. Kueck, L.-M. Bloyet, E. Cassella, T. Zang, F. Schmidt, V. Brusic, G. Tekes, O. Pornillos, S. Whelan, P. D. Bieniasz

This study identifies TRIM69 as an IFN-induced antiviral factor that inhibits VSVIND replication by targeting its phosphoprotein P through a novel transcriptional inhibition mechanism.

bioRxiv · Jun 27, 2019Microbiology