Author

Y. M

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Organism:human· 4 Research Focus:cell type annotation· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

M. K. Jolly· 4 shared S. R· 2 shared A. J. Armstrong· 1 shared A. Paul· 1 shared A. R. Subbalakshmi· 1 shared A. Saha· 1 shared B. C. Miller· 1 shared B. C. Thomas· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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4 shared papersCancer Biology

M. K. Jolly

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4 papers

Recent·Most cited

p53 amyloid pathology with cancer grades and p53 mutations

S. Sengupta, N. Singh, A. Paul, D. Datta, D. Chatterjee, S. Mukherjee, L. Gadhe, J. Devi, Y. M, M. K. Jolly, S. K. Maji

The paper shows p53 amyloid load correlates with cancer grades and shows both mutant and wild-type p53 form amyloids in cancer tissues.

bioRxiv · Jul 14, 2023 · PreprintCancer Biology

A synthetic lethal screen for Snail-induced enzalutamide resistance identifies JAK/STAT signaling as a therapeutic vulnerability in prostate cancer

K. E. Ware, B. C. Thomas, P. Olawuni, M. U. Sheth, N. Hawkey, Y. M, B. C. Miller, K. Vietor, M. K. Jolly, S. Y. Kim, A. J. Armstrong, J. A. Somarelli

The paper identifies JAK/STAT signaling as a therapeutic target for Snail-induced enzalutamide-resistant prostate cancer through synthetic lethal screens and validation in a resistant cell model.

Frontiers in Molecular Biosciences · May 9, 2023Peer-reviewedCancer Biology

PD-L1 activity is associated with partial EMT and metabolic reprogramming in carcinomas

S. Muralidharan, M. Sehgal, S. R, S. Mandal, S. S. Majumdar, Y. M, A. Saha, M. K. Jolly

The paper identifies a link between PD-L1 activity, partial EMT, and metabolic reprogramming in carcinomas using transcriptomic data and single-cell RNA-seq.

bioRxiv · Oct 5, 2022Cancer Biology

The ELF3 transcription factor is associated with an epithelial phenotype and represses epithelial-mesenchymal transition

A. R. Subbalakshmi, S. Sahoo, P. Manjunatha, S. Goyal, V. A. Kasiviswanathan, Y. M, S. R, I. McMullen, J. Somarelli, M. K. Jolly

The paper identifies ELF3 as an epithelial-associated transcription factor that inhibits EMT and stabilizes hybrid E/M phenotypes, with potential prognostic implications in solid tumors.

bioRxiv · Aug 19, 2022Cancer Biology