Y. Sreelakshmi

The paper develops two tomato starter lines with maize transposon elements and transposase to enable large-scale transposon mutagenesis for gene functional validation.

This study introduces a tomato cultivar with extended shelf life, improved taste, and flavor by combining genome-edited ripening mutants with a phototropin1 mutant.

The paper identifies reduced γ-glutamyl hydrolase activity in PKM-1 tomato as a factor contributing to higher folate levels compared to AV.

This study sequenced mutagenized tomato lines to identify mutations, revealing high mutation density and novel insights into EMS mutagenesis patterns and their impact on metabolic pathways.

A tomato trifoliate allele encoding a truncated MYB117 transcription factor alters cellular homeostasis and fortifies γ-aminobutyric acid, folate, and carotenoids in tomato fruits.

The paper shows that PSY2 contributes to abscisic acid formation in ripening tomato fruits by maintaining a parallel carotenogenesis pathway when PSY1 is absent.

This study shows tomato roots sense horizontal/vertical mechanical impedance and modulate metabolite profiles differently in roots and shoots.

A dominant-negative mutation in phototropin1 increases carotenoid content and alters metabolite and protein profiles in tomato fruits.

This study identifies ACS2's role in tomato development beyond fruit ripening by analyzing two contrasting acs2 mutants and their phenotypic and metabolomic differences.

The paper investigates the relationship between polar auxin transport and glutathione levels in tomato mutant pct1-2, finding that GSH levels influence PAT and root elongation.

This study shows NPA stimulates tomato hypocotyl elongation by increasing ethylene and altering indole-3-butyric acid and zeatin levels.

This study identifies NAC-NOR mutations in tomato Penjar accessions that reduce respiration, alter cuticle composition, and increase ABA and sucrose levels, leading to prolonged fruit shelf life.

This study shows that silencing MSH2 in tomato leads to tetraploid meiocytes and abnormal pollen formation due to disrupted meiosis and ploidy stability.