Author

A. Weng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. S. Flozak· 3 shared C. J. Gottardi· 3 shared M. Maciel-Herrerias· 2 shared R. P. Aillon· 2 shared A. Misharin· 1 shared A. V. Misharin· 1 shared C. E. Runyan· 1 shared D. Celli· 1 shared

Activity by Year

2022-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersCell Biology

A. S. Flozak

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Phage administration with repeated intravenous doses leads to faster phage clearance in mammalian hosts

X. Tan, K. Chen, Z. Jiang, Z. Liu, S. Wang, Y. Yong, J. Zhang, S. Yuan, Z. Huang, R. Gao, M. Zhao, A. Weng, Y. Yang, H. Luo, D. Zhang, Y. Ma

This study evaluates phage clearance and host responses to repeated intravenous phage doses in rats and monkeys, finding faster clearance and immune activation with repeated administration.

bioRxiv · Mar 10, 2023 · PreprintPharmacology & Toxicology

Alpha-T-catenin is expressed in peripheral nerves as a constituent of Schwann cell adherens junctions

A. Weng, E. E. Rabin, A. S. Flozak, R. P. Aillon, S. E. Chiarella, C. J. Gottardi

The paper identifies alpha-T-catenin expression in Schwann cell adherens junctions of peripheral nerves and links CTNNA3 to neuroglial functions, potentially explaining its association with non-cardiac diseases.

bioRxiv · Sep 15, 2022Cell Biology

Lung injury induces alveolar type 2 cell hypertrophy and polyploidy with implications for repair and regeneration

A. Weng, M. Maciel-Herrerias, S. J. Watanabe, A. S. Flozak, L. Welch, R. P. Aillon, R. Grant, S. Han, L. Dada, M. HInchcliff, A. Misharin, G. S. Budinger, C. J. Gottardi

This study shows lung injury induces alveolar type 2 cell hypertrophy and polyploidy, which can be reduced by inhibiting the integrated stress response.

American Journal of Respiratory Cell and Molecular Biology · May 1, 2022Peer-reviewedCell Biology

Hypercapnia limits β-catenin mediated alveolar type 2 cell progenitor function by altering Wnt production from adjacent fibroblasts

L. A. Dada, L. C. Welch, N. D. Magnani, Z. Ren, P. L. Brazee, D. Celli, A. S. Flozak, A. Weng, M. Maciel-Herrerias, V. Kryvenko, I. Vadasz, C. E. Runyan, H. Abdala-Valencia, M. Shigemura, S. M. Casalino-Matsuda, , J. I. Sznajder

This study shows hypercapnia inhibits β-catenin signaling in alveolar type 2 cells by altering Wnt production from fibroblasts, impairing lung repair after injury.

bioRxiv · Jan 12, 2022 · PreprintCell Biology