Author

B. Geng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:metabolism & bioenergetics· 1 Assay:protein structure· 1

Frequent Co-Authors

I. Stagljar· 2 shared J. Snider· 2 shared Z. Yao· 2 shared A. Bello· 1 shared A. Casanovas-Massana· 1 shared A. Iwasaki· 1 shared A. Ko· 1 shared A. Ring· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersBiochemistry

I. Stagljar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Omicron Spike Protein Is Vulnerable to Reduction

I. Stagljar, Z. Yao, B. Geng, E. Marcon, S. Pu, H. Tang, J. Merluza, A. Bello, J. Snider, P. Lu, H. Wood

This study shows that Omicron SARS-CoV-2 spike proteins are more susceptible to reduction due to mutations in the receptor binding module, which affects their stability and binding activity.

bioRxiv · Jan 6, 2023Biochemistry

MMD scaffolds ACSL4 and MBOAT7 to promote polyunsaturated phospholipid synthesis and susceptibility to ferroptosis

V. V. Phadnis, J. Snider, V. Wong, K. D. Vaccaro, T. Kunchok, J. Allen, Z. Yao, B. Geng, K. Weiskopf, I. Stagljar, W. S. Henry, R. A. Weinberg

This paper identifies MMD as a scaffold protein that promotes ferroptosis by facilitating the synthesis of arachidonoyl-phospholipids through interactions with ACSL4 and MBOAT7.

bioRxiv · Sep 1, 2022Cell Biology

Exploratory neuroimmune profiling identifies CNS-specific alterations in COVID-19 patients with neurological involvement

E. Song, C. M. Bartley, R. D. Chow, T. Ngo, R. Jiang, C. R. Zamecnik, R. Dandekar, R. Loudermilk, Y. Dai, F. Liu, I. Hawes, B. D. Alvarenga, T. Huynh, L. McAlpine, N.-T. Rahman, , S. F. Farhadian

The paper identifies CNS-specific immune alterations in COVID-19 patients with neurological symptoms, suggesting neuroinvasion and anti-neural autoimmunity as potential causes.

bioRxiv · Sep 11, 2020Immunology